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Spinal Deformity in Familial Dysautonomia Prevalence, and Results of Bracing*
Shlomo Hayek, M.D.†; F. Javier Laplaza, M.D.‡; Felicia B. Axelrod, M.D.§; Stephen W. Burke, M.D.‡
View Disclosures and Other Information
Investigation performed at the Department of Pediatric Orthopaedics, The Hospital for Special Surgery, New York, N.Y.
*No benefits in any form have been received or will be received from a commercial party related directly or indirectly to the subject of this article. No funds were received in support of this study.
†Department of Pediatric Orthopaedics, Tel Aviv Sourasky Medical Center, 6 Weizman Street, Tel Aviv 64239, Israel.
‡Department of Pediatric Orthopaedics, The Hospital for Special Surgery, 535 East 70th Street, New York, N.Y. 10021. E-mail address for F. J. Laplaza: laplazaf@hss.edu. E-mail address for S. W. Burke: burkes@hss.edu.
§Dysautonomia Treatment and Evaluation Center, New York University Medical Center, 530 First Avenue, Suite 9Q, New York, N.Y. 10016. E-mail address: felicia.axelrod@popmail.med.nyu.edu.

The Journal of Bone & Joint Surgery.  2000; 82:1558-1558 
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Abstract

Background: Familial dysautonomia (Riley-Day syndrome) is an autosomal recessive disorder primarily affecting individuals of Ashkenazi Jewish extraction. It affects the autonomic, central, and peripheral nervous systems. Spinal deformity (mainly scoliosis) is the most common orthopaedic problem in patients with familial dysautonomia. The objectives of our study were to document the prevalence of spinal deformity in a referral center for familial dysautonomia and to determine the effectiveness of bracing.

Methods: We performed a retrospective radiographic and clinical study of 123 patients with familial dysautonomia who had survived to the age of twenty years or older.

Results: One hundred and two (83 percent) of the 123 patients had spinal deformity: sixty-nine (56 percent) had scoliosis only, thirty-one (25 percent) had scoliosis as well as kyphosis, and two (2 percent) had kyphosis only. Scoliosis was diagnosed by the age of ten years in sixty-four (52 percent) of the patients. Of the sixty-five patients who were treated with bracing, fifty-eight (89 percent) had progression and twenty-four (37 percent) underwent spinal arthrodesis. No risk factors for the presence or progression of the curves could be found.

Conclusions: The prevalence of spinal deformity in patients with familial dysautonomia who had lived for at least twenty years was found to be 83 percent. By the age of ten years, 52 percent of the patients had scoliosis and 21 percent had kyphosis with or without scoliosis. Bracing was found to be of limited effectiveness as a definitive treatment for spinal deformity. The curve progressed despite bracing in fifty-eight (89 percent) of sixty-five patients.

Figures in this Article
    First described in 194912, familial dysautonomia (Riley-Day syndrome) is an autosomal recessive disorder primarily affecting individuals of Ashkenazi Jewish extraction2,3,13. It has been estimated that one in thirty of these individuals is a carrier (unpublished data). Recently, the gene for familial dysautonomia has been localized in the long arm of chromosome 9q31-q335.
    Neuropathological findings have suggested that familial dysautonomia may be due to incomplete maturation of the unmyelinated neuronal population within the sensory system, the sympathetic system, and parts of the parasympathetic system3,12. The disorder affects the autonomic, central, and peripheral nervous systems. The distinctive clinical characteristics include reduced overflow tearing (alacrima), absence of fungiform papillae on the tongue, impaired perception of pain and temperature, labile blood pressure, and absence of deep tendon reflexes3,6,12. Cardiovascular, pulmonary, and gastrointestinal manifestations may be severe and may lead to early death.
    Familial dysautonomia is associated with a number of orthopaedic problems, including osteopenia, recurrent fractures, aseptic necrosis, and increased external tibial torsion. The most common orthopaedic condition, and the most difficult to treat, is spinal deformity (mainly scoliosis)1,3,10,11,13,15. Estimations of the prevalence of spinal deformity in the population of patients with familial dysautonomia have ranged from 50 to 95 percent4,8,10,14. The aims of our study were to survey a large population of patients with familial dysautonomia in order to obtain an accurate estimation of the prevalence and severity of spinal deformity in this population and to assess the effectiveness of bracing.
     
    Anchor for JumpAnchor for Jump
    +Fig. 1:Kaplan-Meier survivorship curve depicting the percentage of patients without scoliosis at different ages. By the age of ten years, 52 percent (sixty-four) of the 123 patients had been diagnosed with scoliosis. The error bars indicate the 95 percent confidence intervals.
     
    Anchor for JumpAnchor for Jump
    +Fig. 2:Kaplan-Meier survivorship curve depicting the percentage of patients without kyphosis at different ages. By the age of twenty years, 27 percent (thirty-three) of the 123 patients had been diagnosed with kyphosis. The error bars indicate the 95 percent confidence intervals.
    Data on the patients were obtained retrospectively from the files of the Dysautonomia Treatment and Evaluation Center at New York University Medical Center. Of the 480 patients with familial dysautonomia who were listed in the Center's registry, 165 had been born prior to 1976 and had been examined at the Center. Only the 123 patients who had reached the minimum age of twenty years were included in the study. At the time of the survey, seventy-four of these patients were still alive.
    Thirty-three patients were at least twenty years old when they were referred to our institution. Twenty-two of these patients had been diagnosed with spinal deformity before the time of referral; the mean age at the time of diagnosis was nine years (range, four to seventeen years). Although these twenty-two patients had been diagnosed at another institution, the compilation of data for the study began at the time of the diagnosis. The magnitude of the initial curve could not be determined for nine of these patients (six female and three male) because the early radiographs could not be obtained. According to the records, the deformity had been diagnosed at a mean age of eight years and all nine patients had been treated with observation only.
    Data on the spinal curves were obtained from computer records and radiographs. For some patients, only serial chest radiographs were available, but these radiographs were adequate for the detection and measurement of thoracic spinal deformity. Scoliosis was defined as a Cobb angle7 of 11 degrees or more in the coronal plane, and kyphosis was defined as an angle of 41 degrees or more in the sagittal plane. Scoliosis was classified as mild (11 to 20 degrees), moderate (21 to 40 degrees), or severe (41 degrees or more). In the present study, kyphosis was limited to the thoracic spine and, in most patients, the apex of the kyphosis was at the fifth thoracic level.
    In addition to the measurement of spinal curvature, we also evaluated the type, duration, and effect of treatment as well as the age at which treatment was initiated.

    Statistical Analysis

    A statistician evaluated all of our data. Variables were tested with one-way analysis of variance followed by Tukey's HSD (honestly significant difference) test for multiple comparisons. A p value of less than 0.05 was considered significant.

    Survivorship Analysis

    Kaplan-Meier survivorship analysis9 was performed to estimate the probability of development of scoliosis or kyphosis. The end point was defined as the age at which the diagnosis of spinal deformity was made. Therefore, in our study the term "survivorship" does not refer to the patients' being alive but to their having not been diagnosed with spinal deformity.
    There were sixty-nine female patients and fifty-four male patients. Most of the patients were first seen at the Dysautonomia Treatment and Evaluation Center during the first year of life, but some were not referred until adolescence or even adulthood. The mean age at the time of the initial evaluation at our institution was sixteen years (range, six months to forty-four years), and the mean duration of follow-up was 16.5 years (range, one to twenty-six years).

    Epidemiology of the Spinal Deformity

    Of the 123 patients with familial dysautonomia, 102 (83 percent) had been diagnosed with spinal deformity and twenty-one (17 percent) had not. Sixty-nine patients (56 percent) had scoliosis only, thirty-one (25 percent) had scoliosis as well as kyphosis, and only two (2 percent) had kyphosis without scoliosis. The mean scoliotic curve was 30 degrees, and the mean kyphotic curve was 79 degrees. Scoliosis was diagnosed at a mean age of nine years (range, six months to seventeen years), and kyphosis was diagnosed at a mean age of eight years (range, three to sixteen years).
    Both genders were affected equally. The male:female ratio in the population of patients who had scoliosis was 0.82 (forty-five male to fifty-five female patients). This was similar to the ratio in the overall study population, which was 0.78 (fifty-four male to sixty-nine female patients).
    Kaplan-Meier analysis, with the end point defined as the age at which the diagnosis of spinal deformity was made, revealed that sixty-four (52 percent) of the 123 patients with familial dysautonomia had scoliosis by the age of ten years (Fig. 1) and twenty-six (21 percent) had kyphosis (with or without scoliosis) by the same age (Fig. 2).

    Type and Severity of Scoliosis

    Unlike idiopathic scoliosis, in which most curves are right thoracic and left lumbar, forty of the 100 scoliotic curves in our series were left thoracic. Nine were thoracic curves only, thirty were associated with an additional right lumbar curve, and one was a left thoracolumbar curve. At the time of presentation, eighteen of the 100 curves were rated as mild; twenty-two, as moderate; and sixty, as severe.

    Treatment

    The results of conservative treatment were assessed in ninety-four of the 102 patients with spinal deformity. These ninety-four patients were divided into two groups: the observation group (twenty-nine patients) and the bracing group (sixty-five patients). The remaining eight patients, who had a mean curve of 70 degrees, had surgery as the primary treatment at a mean age of eighteen years.

    Observation Group

    Eleven of the twenty-nine patients in the observation group had a severe scoliotic curve. In nine of these eleven patients, the initial curve could not be documented because they had been born prior to 1953 and had first been seen at our center after the age of twenty years. In the other two patients, the curve had progressed from 19 to 55 degrees over ten years and from 70 to 106 degrees over eleven years. The pulmonary status of the eleven patients with a severe curve was believed to be so frail that any type of treatment was contraindicated. In the eighteen patients with a mild-to-moderate scoliotic curve, the mean curve progressed from 14 to 22 degrees over seventeen years, for a mean progression of 0.5 degree per year.

    Bracing Group

    Sixty-five patients were treated with bracing at a mean age (and standard deviation) of 12 ± 4 years (range, three to twenty years). The brace was worn for a mean of five years (range, six months to fourteen years). The Milwaukee brace was worn by fifty-six patients, and a thoracolumbosacral orthosis was worn by nine. Initially, the brace was to be worn full time, but we noted exacerbation of gastroesophageal reflux and aspiration when the brace was worn at night. This was probably because the patients were in a recumbent position, they were being fed at night through a gastrostomy tube, and their gag reflex was already compromised. Therefore, bracing was discontinued at night. Currently, our patients (except for those with excessive kyphosis of more than 50 degrees) are treated with a thermoplastic thoracolumbosacral orthosis.
    Fifty-eight (89 percent) of the sixty-five patients had progression of the curve despite bracing. The thoracic curves progressed a mean of 5 degrees per year (from 25 to 50 degrees), the lumbar curves progressed a mean of 4 degrees per year (from 19 to 38 degrees), and kyphosis increased a mean of 5 degrees per year (from 53 to 77 degrees). In contrast, seven (11 percent) of the sixty-five patients who wore a brace did not have progression of the curve. In these seven patients, the mean thoracic curve was 21 degrees (range, 11 to 44 degrees) at the beginning of bracing and 21 degrees (range, 9 to 49 degrees) at the end of bracing. In this group, bracing was started at a mean age of twelve years and was continued for a mean of four years; this was not significantly different from the duration of bracing in the group of patients who had progression. We found no association between the magnitude of the curve and the change that occurred during bracing.
    Of the fifty-eight patients for whom bracing had failed, twenty-four underwent spinal arthrodesis. The remaining thirty-four patients were not operated on because of their precarious cardiopulmonary status, because they had been seen for consultation only, or because they did not want an operation.
    Because the data in our study originated from the Dysautonomia Treatment and Evaluation Center (a comprehensive care facility for patients with this disorder) and because the sample size was larger than it has been in previous studies, we believe that we are able to provide an extensive analysis of the prevalence of spinal deformity in this population. Other retrospective studies on the prevalence of scoliosis in patients with familial dysautonomia were from orthopaedic referral centers. In 1971, Yoslow et al.15 reported on sixty-five patients with familial dysautonomia who ranged in age from nine months to twenty-nine years. Thirty-nine (60 percent) of the patients were found to have scoliosis. Five patients had severe scoliosis measuring over 50 degrees, but none were treated operatively. In 1987, Albanese and Bobechko1 reviewed the cases of sixteen patients with familial dysautonomia. Nine of the patients had spinal deformity: two had scoliosis, four had scoliosis and kyphosis, and three had predominantly kyphosis. Seven patients were treated operatively with spinal arthrodesis and Harrington compression instrumentation following the failure of bracing. In 1995, Rubery et al.14 described their experience with twenty-two patients who had operative treatment for the correction of spinal deformity. In that study, it was estimated that progressive scoliosis would develop in 95 percent of patients with familial dysautonomia and that kyphosis would develop in 53 percent.
    In an attempt to define the probability of a spinal deformity developing during growth in patients with familial dysautonomia, we reviewed the data on all such patients who had reached the age of twenty years or more. We found that 102 (83 percent) of the 123 patients had a spinal deformity: sixty-nine (56 percent) had scoliosis only, thirty-one (25 percent) had scoliosis associated with kyphosis, and two (2 percent) had kyphosis only. Forty of the 100 scoliotic curves were convex to the left. This is in agreement with the findings reported in the 1997 study by Kaplan et al.10, in which 51 percent (twenty-six) of the fifty-one curves in patients with familial dysautonomia were convex to the left.
    These deformities develop early and progress rapidly during the early years of life. In our study, scoliosis had been diagnosed in half of the patients by the age of ten years (Fig. 1).
    A limitation of our study was the lack of access to the initial radiographs of nine untreated patients who already had severe curves at the time of presentation to our institution. The progression of these curves could not be assessed.
    The etiology of spinal deformity in patients with familial dysautonomia is not clear. The curves tend to be severe and progressive, resembling deformities found in patients with other neuromuscular diseases8.
    The treatment of spinal deformity in these patients is difficult and frustrating. As the life expectancy of patients with familial dysautonomia has increased, such treatment has become a major issue related to their medical care. In 1970, Brunt and McKusick6 predicted that only 50 percent of all children born with familial dysautonomia would survive beyond the age of five years. In our study, 123 (75 percent) of 165 patients had survived to the age of at least twenty years, with the oldest patient being fifty-one years old.
    In the present study, bracing was found to be of limited effectiveness for the treatment of spinal deformity in patients with familial dysautonomia. Even with bracing, most of the curves continued to progress quite rapidly, at a rate of about 4 to 5 degrees per year. Bracing prevented progression of the curve in only seven (11 percent) of sixty-five patients. Most of the curves that did not progress had been mild (mean, 21 degrees) at the onset of bracing. We found no other variable to be predictive of which curves would stabilize. These findings are in agreement with those of Kaplan et al.10, who reported a progression rate of 2.8 degrees for scoliosis and 3.5 degrees for kyphosis. In their series, all ten patients who were managed with a Milwaukee brace had progression of the curve and three of the seven patients who were managed with a thoracolumbosacral orthosis needed spinal arthrodesis. It was not clear whether the curves in the other four patients had stabilized or whether they simply had not progressed enough to necessitate surgery.
    Forty of the 100 scoliotic curves in our study were left thoracic. This is in contrast to curves in patients with adolescent idiopathic scoliosis, which are usually right thoracic with the apex at the seventh or eighth thoracic vertebra.
    Our protocol for the treatment of spinal deformity in immature patients with familial dysautonomia calls for the use of a custom-molded spinal orthosis for curves of at least 20 degrees that have progressed a total of 5 degrees or more since the initial evaluation. We believe that a frank discussion with the family should be carried out in order to outline the poor response to bracing; however, we continue to recommend bracing as the last resort in an attempt to avoid surgery and its complications in this medically fragile population. We still believe that bracing may help to slow the progression of mild curves. Although bracing was initially used in a small number of skeletally mature patients because it was thought to be the only option that would stop curve progression, this is not currently done. When a curve progresses to 45 degrees or more in an immature patient, the patient becomes a candidate for spinal arthrodesis with instrumentation.
    In summary, the prevalence of spinal deformity in patients with familial dysautonomia who had lived for at least twenty years was 83 percent. By the age of ten years, 52 percent of the patients had scoliosis and 21 percent had kyphosis with or without scoliosis. Bracing was found to be of limited effectiveness as a definitive treatment for spinal deformity. With the increasing longevity of these patients, aggressive operative treatment may provide the only means of controlling severe deformities despite the high risk of serious complications. We could not establish a means of predicting the progression of a given curve with our data. Improved technology and increased knowledge of the disease will facilitate our approach to the treatment of patients with familial dysautonomia.
    Albanese, S. A., and Bobechko, W. P.: Spine deformity in familial dysautonomia (Riley-Day syndrome). J. Pediat. Orthop.,7: 179-183, 1987.7179  1987 
     
    Axelrod, F. B., and Abularrage, J. J.: Familial dysautonomia: a prospective study of survival. J. Pediat.,101: 234-236, 1982.101234  1982  [PubMed]
     
    Axelrod, F. B.: Familial dysautonomia. In Disorders of the Autonomic Nervous System, pp. 217-231. Edited by D. Robertson and I. Biaggioni. From the series The Autonomic Nervous System (vol. 5), edited by G. Burnstock. Luxembourg, Harwood Academic Publishers, 1995. 
     
    Bassett, G. S.: Other neuromuscular disorders. In The Pediatric Spine: Principles and Practice, edited by S. L. Weinstein. Vol. 2, pp. 1089-1102. New York, Raven Press, 1994. 
     
    Blumenfeld, A.; Slaugenhaupt, S. A.; Axelrod, F. B.; Lucente, D. E.; Maayan, C.; Liebert, C. B.; Ozelius, L. J.; Trofatter, J. A.; Haines, J. L.; Breakefield, X. O.; and Gusella, J. F.: Localization of the gene for familial dysautonomia on chromosome 9 and definition of DNA markers for genetic diagnosis. Nature Genet.,4: 160-164, 1993.4160  1993  [PubMed]
     
    Brunt, P. W., and McKusick, V. A.: Familial dysautonomia. A report of genetic and clinical studies, with a review of the literature. Medicine, ,49: 343-374, 1970.49343  1970 
     
    Cobb, J. R.: Outline for the study of scoliosis. In Instructional Course Lectures, American Academy of Orthopaedic Surgeons. Vol. 5, pp. 261-275. Ann Arbor, J. W. Edwards, 1948. 
     
    Hensinger, R. N., and MacEwen, G. D.: Spinal deformity associated with heritable neurological conditions: spinal muscular atrophy, Friedreich's ataxia, familial dysautonomia, and Charcot-Marie-Tooth disease. J. Bone and Joint Surg.,58-A: 13-24, Jan 1976.58-A13  1976 
     
    Kaplan, E. L., and Meier, P.: Nonparametric estimation from incomplete observations. J. Am. Statist. Assn.,53: 457-481, 1958.53457  1958 
     
    Kaplan, L.; Margulies, J. Y.; Kadari, A.; Floman, Y.; and Robin, G. C.: Aspects of spinal deformity in familial dysautonomia (Riley-Day syndrome). European Spine J.,6: 33-38, 1997.633  1997 
     
    Levine, D. B.: Orthopaedic aspects of familial dysautonomia. In Scoliosis and Muscle. Proceedings of a Fourth Symposium held at The Cardiothoracic Institute, Brompton Hospital, London, on 14th and 15th November 1973, pp. 143-150. Edited by P. A. Zorab. Philadelphia, J. B. Lippincott, 1974. 
     
    Riley, C. M.; Day, R. L.; Greeley, D. M.; and Langford, W. S.: Central autonomic dysfunction with defective lacrimation: report of five cases. Pediatrics,3: 468-478, 1949.3468  1949  [PubMed]
     
    Robin, G. C.: Scoliosis in familial dysautonomia. Bull. Hosp. Joint Dis.,44: 16-26, 1984.4416  1984 
     
    Rubery, P. T.; Spielman, J. H.; Hester, P.; Axelrod, F. B.; Burke, S. W.; and Levine, D. B.: Scoliosis in familial dysautonomia. Operative treatment. J. Bone and Joint Surg.,77-A: 1362-1369, Sept 1995.77-A1362  1995 
     
    Yoslow, W.; Becker, M. H.; Bartels, J.; and Thompson, W. A. L.: Orthopaedic defects in familial dysautonomia. A review of sixty-five cases. J. Bone and Joint Surg.,,53-A: 1541-1550, Dec 1971.53-A1541  1971 
     

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    Anchor for JumpAnchor for Jump
    +Fig. 1:Kaplan-Meier survivorship curve depicting the percentage of patients without scoliosis at different ages. By the age of ten years, 52 percent (sixty-four) of the 123 patients had been diagnosed with scoliosis. The error bars indicate the 95 percent confidence intervals.
    Anchor for JumpAnchor for Jump
    +Fig. 2:Kaplan-Meier survivorship curve depicting the percentage of patients without kyphosis at different ages. By the age of twenty years, 27 percent (thirty-three) of the 123 patients had been diagnosed with kyphosis. The error bars indicate the 95 percent confidence intervals.
    Albanese, S. A., and Bobechko, W. P.: Spine deformity in familial dysautonomia (Riley-Day syndrome). J. Pediat. Orthop.,7: 179-183, 1987.7179  1987 
     
    Axelrod, F. B., and Abularrage, J. J.: Familial dysautonomia: a prospective study of survival. J. Pediat.,101: 234-236, 1982.101234  1982  [PubMed]
     
    Axelrod, F. B.: Familial dysautonomia. In Disorders of the Autonomic Nervous System, pp. 217-231. Edited by D. Robertson and I. Biaggioni. From the series The Autonomic Nervous System (vol. 5), edited by G. Burnstock. Luxembourg, Harwood Academic Publishers, 1995. 
     
    Bassett, G. S.: Other neuromuscular disorders. In The Pediatric Spine: Principles and Practice, edited by S. L. Weinstein. Vol. 2, pp. 1089-1102. New York, Raven Press, 1994. 
     
    Blumenfeld, A.; Slaugenhaupt, S. A.; Axelrod, F. B.; Lucente, D. E.; Maayan, C.; Liebert, C. B.; Ozelius, L. J.; Trofatter, J. A.; Haines, J. L.; Breakefield, X. O.; and Gusella, J. F.: Localization of the gene for familial dysautonomia on chromosome 9 and definition of DNA markers for genetic diagnosis. Nature Genet.,4: 160-164, 1993.4160  1993  [PubMed]
     
    Brunt, P. W., and McKusick, V. A.: Familial dysautonomia. A report of genetic and clinical studies, with a review of the literature. Medicine, ,49: 343-374, 1970.49343  1970 
     
    Cobb, J. R.: Outline for the study of scoliosis. In Instructional Course Lectures, American Academy of Orthopaedic Surgeons. Vol. 5, pp. 261-275. Ann Arbor, J. W. Edwards, 1948. 
     
    Hensinger, R. N., and MacEwen, G. D.: Spinal deformity associated with heritable neurological conditions: spinal muscular atrophy, Friedreich's ataxia, familial dysautonomia, and Charcot-Marie-Tooth disease. J. Bone and Joint Surg.,58-A: 13-24, Jan 1976.58-A13  1976 
     
    Kaplan, E. L., and Meier, P.: Nonparametric estimation from incomplete observations. J. Am. Statist. Assn.,53: 457-481, 1958.53457  1958 
     
    Kaplan, L.; Margulies, J. Y.; Kadari, A.; Floman, Y.; and Robin, G. C.: Aspects of spinal deformity in familial dysautonomia (Riley-Day syndrome). European Spine J.,6: 33-38, 1997.633  1997 
     
    Levine, D. B.: Orthopaedic aspects of familial dysautonomia. In Scoliosis and Muscle. Proceedings of a Fourth Symposium held at The Cardiothoracic Institute, Brompton Hospital, London, on 14th and 15th November 1973, pp. 143-150. Edited by P. A. Zorab. Philadelphia, J. B. Lippincott, 1974. 
     
    Riley, C. M.; Day, R. L.; Greeley, D. M.; and Langford, W. S.: Central autonomic dysfunction with defective lacrimation: report of five cases. Pediatrics,3: 468-478, 1949.3468  1949  [PubMed]
     
    Robin, G. C.: Scoliosis in familial dysautonomia. Bull. Hosp. Joint Dis.,44: 16-26, 1984.4416  1984 
     
    Rubery, P. T.; Spielman, J. H.; Hester, P.; Axelrod, F. B.; Burke, S. W.; and Levine, D. B.: Scoliosis in familial dysautonomia. Operative treatment. J. Bone and Joint Surg.,77-A: 1362-1369, Sept 1995.77-A1362  1995 
     
    Yoslow, W.; Becker, M. H.; Bartels, J.; and Thompson, W. A. L.: Orthopaedic defects in familial dysautonomia. A review of sixty-five cases. J. Bone and Joint Surg.,,53-A: 1541-1550, Dec 1971.53-A1541  1971 
     
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