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Mechanical Prophylaxis Against Deep-Vein Thrombosis After Pelvic and Acetabular Fractures
James P. Stannard, MD; Reneé S. Riley, MD; Michelle D. McClenney, RN; Robert R. Lopez-Ben, MD; David A. Volgas, MD; Jorge E. Alonso, MD
View Disclosures and Other Information
Investigation performed at the University of Alabama Hospital, Birmingham, Alabama
James P. Stannard, MD
Reneé S. Riley, MD
Michelle D. McClenney, RN
Robert R. Lopez-Ben, MD
David A. Volgas, MD
Jorge E. Alonso, MD
Division of Orthopaedic Surgery, University of Alabama Hospital, 509 Medical Education Building, 1813 6th Avenue South, Birmingham, AL 35294-3295. E-mail address for J.P. Stannard: james.stannard@ortho.uab.edu

Although none of the authors has received or will receive benefits for personal or professional use from a commercial party related directly or indirectly to the subject of this article, benefits have been or will be received, but are directed solely to a research fund, foundation, educational institution, or other nonprofit organization with which one or more of the authors is associated. Funds were received in total or partial support of the research or clinical study presented in this article. The funding source was NuTech, Kinetic Concepts, Incorporated.

The Journal of Bone & Joint Surgery.  2001; 83:1047-1051 
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Abstract

Background: Deep-vein thrombosis is a common complication following pelvic and acetabular fractures. The hypothesis of this study was that pulsatile mechanical compression is superior to standard sequential mechanical compression for decreasing the prevalence of deep-vein thrombosis in patients with pelvic or acetabular fracture.

Methods: A prospective, randomized, blinded study of two methods of mechanical prophylaxis against deep-vein thrombosis was conducted. One hundred and seven patients were randomized into either Group A (fifty-four patients), in which a thigh-calf low-pressure sequential-compression device was used, or Group B (fifty-three patients), in which a calf-foot high-pressure pulsatile-compression pump was used. All patients underwent duplex ultrasonography and magnetic resonance venography. The two groups were comparable with regard to demographics, fracture type, fracture treatment, time from the injury to the prophylaxis, and patient compliance.

Results: Deep-vein thrombosis developed in ten patients (19%) in Group A, with seven (13%) having a large or occlusive clot and one (2%) having a documented pulmonary embolism. Deep-vein thrombosis developed in five patients (9%) in Group B, with two (4%) having a large or occlusive clot and none having a documented pulmonary embolism. Nine of the nineteen detected thromboses were in the deep pelvic veins. The difference in the prevalence of large or occlusive clots between the two groups demonstrated a trend but, with the numbers available, was not significant (p = 0.16). Increased patient age and the time elapsed from the injury to the surgery were found to be associated with higher rates of thrombosis.

Conclusions: Pulsatile compression was associated with fewer deep-vein thromboses than was standard compression, with the difference representing a trend but not reaching significance with the number of patients studied.

Figures in this Article
    Venous thromboembolic disease is a frequent complication following blunt trauma1-6. Patients with pelvic or acetabular fracture are a subpopulation of patients with multiple trauma who have been identified as being at high risk for the development of deep-vein thrombosis4. Pulmonary embolism is the most common cause of deaths occurring more than seven days after traumatic injury7.
    Because of the risk of internal bleeding, especially in patients with a head injury, several authors have expressed concerns regarding aggressive anticoagulation as primary prophylaxis for patients who have sustained traumatic injuries7-10. Some authors have utilized inferior vena cava filters as either prophylaxis against or treatment of venous thromboembolic disease2,7, but others have expressed major concerns regarding the long-term consequences of the use of such devices11. Mechanical compression is an alternative to pharmacological prophylaxis that avoids the risk of bleeding complications.
    Mechanical compression to prevent deep-vein thrombosis is currently available in two forms: thigh-calf low-pressure sequential-compression devices, and high-pressure pulsatile-compression devices that are available with either a calf-foot or a foot-only wrap. Low-pressure sequential-compression devices inflate the thigh and calf chambers sequentially to a pressure of approximately 45 mm Hg. Combination pulsatile-compression devices utilize calf and foot chambers and inflate to a pressure of approximately 160 mm Hg. These pumps differ from the standard foot pumps because they have two bladders, one of which inflates over the calf and one of which inflates over the foot. The pulsatile pumps inflate for only two to three seconds in each twenty-second cycle. There have been several recent articles describing the use of these two forms of mechanical compression to prevent deep-vein thrombosis in patients who have sustained traumatic injuries3,9,10,12.
    The hypothesis of our study was that pulsatile compression is more effective than sequential compression in preventing deep-vein thrombosis in patients with pelvic or acetabular fracture requiring internal fixation.
    This study was designed as a prospective, randomized, and blinded evaluation of sequential and pulsatile mechanical compression devices. All patients admitted to the University of Alabama Hospital with a pelvic or acetabular fracture due to blunt trauma between December 1, 1997, and May 1, 1999, were evaluated for possible inclusion in the study. Inclusion criteria included blunt trauma causing a pelvic or acetabular fracture with a pattern requiring surgical fixation, an age of at least sixteen years, and an ability and willingness to comply with both the mechanical prophylaxis protocol and the screening studies for deep-vein thrombosis. Exclusion criteria included a history of venous thromboembolic disease, initiation of mechanical compression more than seventy-two hours following the injury, a body habitus or weight that made it difficult for the patient to fit in the magnetic resonance imaging scanner, or a stable injury that did not require surgical treatment. The institutional review board of the University of Alabama Hospital approved this study.
    Patients who agreed to participate in this study were randomized into one of two treatment groups for prophylaxis against deep-vein thrombosis, and informed consent was obtained. Randomization was accomplished by using a computer-generated randomization table. Patients randomized into Group A were treated bilaterally with a thigh-calf sequential-compression device (Kendall SCD; Kendall, Mansfield, Massachusetts). Patients randomized into Group B were treated bilaterally with a combination sequential pump that covers the calf and foot (PlexiPulse; NuTech, Kinetic Concepts, San Antonio, Texas). Patients were treated with the mechanical prophylaxis as soon as possible following admission to the trauma service. The patients and the nursing staff were encouraged to utilize the pumps for the maximum number of hours possible per day, with removal allowed only for nursing care and physical therapy. The nursing staff was instructed to ensure proper application of wraps and to document the number of hours of wear on a data sheet. No patient who remained in the study received any form of pharmacological prophylaxis, and no patient withdrew from the study voluntarily because of an inability to tolerate the pumps.
    Two different screening studies were performed to evaluate patients for the presence of deep-vein thrombosis. The studies were done within twenty-four hours prior to the patient’s discharge, or earlier if the patient demonstrated any signs or symptoms consistent with venous thromboembolic disease. All screening studies were performed bilaterally. Duplex ultrasound examinations were performed with an Acuson 128 XP or Sequoia System (Mountain View, California) or an ATL 3000HDI machine (Advanced Technology Laboratories, Bothell, Washington) and high-resolution linear transducers with use of color and spectral Doppler vessel interrogation. Standard compression and flow augmentation techniques were utilized in the lower extremities from the groin to the popliteal fossa. Ultrasound criteria for deep-vein thrombosis included visualization of an intraluminal thrombus, loss of vessel compressibility, and decreased blood flow. Magnetic resonance venograms of the pelvis and lower extremities were made with a Signa 1.5-tesla magnet (General Electric Medical Systems, Milwaukee, Wisconsin) and use of two-dimensional time-of-flight sequences (SPGR [spoiled gradient-recalled acquisition in the steady state]; repetition time, 47 msec; echo time, 10.4 msec; 2-mm-thick axial images without gap; 256 ¥ 128 matrix; and one excitation) with saturation of arterial signal. Two musculoskeletal radiologists reviewed the coronal reformatted multiple-intensity projections and source axial images. Discrepancies between the readings were resolved by the radiologists reviewing the study in question together and coming to a consensus.
    Magnetic resonance venogram (MRV) criteria for deep-vein thrombosis included intraluminal signal-flow voids with abrupt narrowing or termination of luminal signal that could not be accounted for by flow or magnetic susceptibility artifacts from internal fixation devices. All screening studies were interpreted with the radiologists blinded regarding the type of prophylaxis against deep-vein thrombosis and the result of the other study. When the two screening tests had conflicting results, the magnetic resonance venography was judged to be valid if the deep-vein thrombosis was located in the pelvis. If not, the staff radiologists settled the discrepancy by reviewing both studies and ordering a repeat ultrasound or venogram if necessary. The end point of the study was when the two imaging studies had been performed to evaluate for deep-vein thrombosis, although patients were followed clinically for the development of deep-vein thrombosis and pulmonary embolism on an outpatient basis. A documented pulmonary embolism was accepted as evidence of deep-vein thrombosis without a corroborating magnetic resonance venogram because of the difficulty involved in the performance of magnetic resonance venography on critically ill patients.
    Data collected included the presence or absence of deep-vein thrombosis and pulmonary embolism, the type and location of deep-vein thrombosis, the time of commencement of mechanical compression, the time between the injury and the surgical stabilization, the number of hours that the pump was worn, and the time between the injury and the screening studies. Because some authors had suggested that magnetic resonance venography may detect deep-vein thromboses that are small and clinically irrelevant13, we documented whether a clot was occlusive or nonocclusive and, if it was nonocclusive, whether it was greater or less than 2 cm in size. Additional data included the use of femoral intravenous lines, the AO classification14 of the fracture, the surgical approach, the injury severity score15, and the presence of additional skeletal injuries.
    A number of different statistical tests were used to analyze the data in this study. The Fisher exact and chi-square tests were employed to determine the significance of the difference between the pulsatile and sequential forms of mechanical prophylaxis. Analysis of variance was used to determine the significance of the difference, with regard to age, injury severity, and time to surgery, between patients with and those without deep-vein thrombosis.
    All patients admitted to this study had sustained a pelvic or acetabular fracture due to blunt trauma and required surgical fixation. The average injury severity score was 19.8 points (range, 9 to 59 points) in Group A and 16.1 points (range, 9 to 50 points) in Group B. The screening tests were done at an average of 6.0 days following the surgery and 10.8 days following the injury in Group A and at an average of 6.5 days following the surgery and 10.8 days following the injury in Group B. There was no significant difference between the two groups with respect to the injury severity score or the time to the screening studies (p > 0.05). There was also no significant difference with regard to gender, weight, AO classification of the fracture, associated long-bone fractures, number of patients with femoral vein cannulation, or surgical approach. The patients in Group A utilized the pumps for an average of 20.8 hours (range, four to twenty-four hours) per day compared with 21.3 hours (range, seven to twenty-four hours) per day in Group B. Because our data regarding patient compliance were incomplete, no statistical analyses correlating usage of the pump with the development of clots were performed.
    Thirty-three patients who were initially enrolled in the study did not complete it. The reasons for withdrawal from the study included claustrophobia (six patients); death (six) (no patient died because of a thromboembolic event); refusal to undergo magnetic resonance venography (five); inadvertent initiation of anticoagulation by another service (five); discharge before the appropriate studies had been performed (three); inability of the patient to fit in the magnetic resonance scanner (two); inability of the patient to remain immobile during the magnetic resonance imaging secondary to a closed head injury (two); prior venous thromboembolic disease missed on the initial screening (two); inadvertent switching of the pump types (one); and pregnancy (one). The demographics of these patients were not significantly different from those of the patients who successfully completed the study. The patients who withdrew had a total of three deep-vein thromboses, which were not included in the statistical analysis.
    One hundred and seven patients completed the protocol and had satisfactory screening studies. Deep-vein thrombosis developed in ten (19%) of the fifty-four patients who completed the protocol in Group A; seven (13%) had a large or occlusive clot and one had a pulmonary embolus. Deep-vein thrombosis developed in five (9%) of the fifty-three patients who completed the study in Group B; two (4%) had a large or occlusive clot and none had a pulmonary embolus. There were no deaths due to venous thromboembolic disease during the study. The overall incidence of deep-vein thrombosis in the study was fifteen (14%) of 107. The difference in the rate of deep-vein thrombosis between Group A and Group B was not significant (p = 0.265), although there was a trend toward more large or occlusive clots in Group A than in Group B (p = 0.16) .
    The primary anatomic locations of the deep-vein thromboses in the fourteen patients in whom the locations were identified included the pelvis (six patients), the thigh (seven), and the calf (one). Four patients had thrombosis in more than one vein. Three thromboses involved the common iliac vein; five, the external iliac vein; one, the internal iliac vein; nine, the common femoral vein; and one, the popliteal vein.
    Analysis of the pooled data from both groups demonstrated a significant difference, in terms of age and time to surgery, between the patients with occlusive deep-vein thrombosis and those with no deep-vein thrombosis or with nonocclusive deep-vein thrombosis. The average age was forty-six years for the patients with occlusive deep-vein thrombosis compared with thirty-five years for those with no deep-vein thrombosis and twenty-six years for those with nonocclusive deep-vein thrombosis (p = 0.03, analysis of variance). The average time between the injury and the surgery was ten days for the patients with occlusive deep-vein thrombosis compared with five days for those with no deep-vein thrombosis and four days for those with nonocclusive deep-vein thrombosis (p = 0.0004, analysis of variance). Patients with an occlusive clot had an average injury severity score of 23.0 points compared with 13.7 and 17.7 points for the patients with nonocclusive and no deep-vein thrombosis, respectively. The difference between the groups represents a trend but was not significant with our sample size (p = 0.19).
    Multivariate analysis of variance demonstrated a trend toward the development of deep-vein thrombosis with the Kocher-Langenbeck and extensile or combined approaches. All nine occlusive deep-vein thromboses, and thirteen of the fifteen deep-vein thromboses detected overall, were in patients treated with one of these approaches. One nonocclusive deep-vein thrombosis was associated with an ilioinguinal approach, and one was associated with a Pfannenstiel approach.
    In one patient in Group A, a late deep-vein thrombosis developed following negative screening studies. Leg pain and increased swelling developed two months following surgery, at which time the patient had a positive ultrasound examination documenting the deep-vein thrombosis. Screening studies were not routinely performed on patients following discharge from the hospital.
    Deep-vein thrombosis has been recognized as a major cause of morbidity and mortality following major blunt trauma3-6,9,10,12,16-20. Geerts et al. documented a 61% prevalence of deep-vein thrombosis following pelvic fractures in patients who had received no prophylaxis against it4. While it is not clear that all of their patients had fractures requiring surgical stabilization, their series was clearly composed of patients with severe traumatic injuries. Using contrast venography, Geerts et al. also documented an 80% prevalence of deep-vein thrombosis associated with femoral fractures. In patients receiving prophylaxis following major trauma, the prevalence of deep-vein thrombosis has ranged from 2%16 to 33%18, depending on the patient population studied, the type of screening study, and the type of prophylaxis against the deep-vein thrombosis.
    Mechanical prophylaxis against deep-vein thrombosis provides protection without increasing the risk of blood loss. Mechanical devices work by improving venous blood flow as well as by stimulating endogenous fibrinolytic activity21. The effectiveness of mechanical prophylaxis in patients undergoing elective total joint replacement has been well documented. However, the literature regarding mechanical prophylaxis after multiple trauma is sparse3,9,12, with reported rates of deep-vein thrombosis ranging from 4% to 21%. Spain et al. evaluated 184 consecutive patients in a retrospective study comparing sequential and pulsatile pumps10. Sequential compression was used when possible, and pulsatile compression was used when the patient had a lower-extremity fracture or another contraindication to sequential compression. The authors found a 7% prevalence of deep-vein thrombosis with sequential compression and a 3% prevalence with pulsatile compression. The retrospective, nonrandomized design as well as the variety of injury types were weaknesses of that study.
    Ultrasound is notoriously poor at detecting pelvic clots. This problem is exacerbated in the setting of trauma with the presence of a pelvic hematoma. Ascending venography is invasive and also lacks sensitivity for detecting pelvic deep-vein thromboses. Montgomery et al. documented a false-negative rate of 58% for ascending venography compared with magnetic resonance venography18. A major difficulty in evaluating these different studies is that it is not clear what test, if any, represents a "gold standard" for detecting pelvic deep-vein thromboses. Ascending venography clearly is not accurate6,18. While Stover et al.13 advocated direct cannulation venography, other authors have noted that it is not accurate for the internal iliac system6,18. The accuracy of magnetic resonance venography depends upon experienced interpretation by radiologists familiar with the technique in patients with traumatic injury. It has a high sensitivity and may detect clots that are small and not at risk for embolization. We attempted to address this problem by presenting our results in terms of the total number of deep-vein thromboses as well as the number of large (2-cm) or occlusive deep-vein thromboses. We are not certain of the importance of small nonocclusive deep-vein thromboses in the pelvic veins; however, because of their proximal location and the potential for clot propagation, we elected to treat these patients for deep-vein thrombosis.
    Magnetic resonance venography has the major advantage of being able to detect thromboses in the pelvic veins as well as in the thigh. In our study, six of fourteen patients with identified deep-vein thrombosis had involvement of a pelvic vein, a finding that is consistent with the results reported by Montgomery et al.18. Numerous previous studies have documented pulmonary embolism despite normal findings on ultrasound or ascending venography5,6,16,18. A probable source for these emboli is the pelvic venous system.
    The primary weakness of our study is that the sample size was not adequate to prove superiority of one type of mechanical prophylaxis over the other. Statistical evaluation demonstrated that approximately 350 patients would be necessary if current trends continued. Another weakness of the study centers on the reliability of the data on patient compliance. Some patients find mechanical pumps uncomfortable, and compliance by patients and by hospital personnel remains an acknowledged problem in this and other studies8.
    This study confirms that patient age is a critical risk factor in the development of deep-vein thrombosis following trauma. The patients in whom occlusive deep-vein thrombosis developed were an average of eleven years older than those in whom it did not develop. There was also a significant association between the time to surgery and the development of thrombosis, with the delay being more than twice as long (9.8 compared with 4.7 days) for patients in whom an occlusive clot developed compared with those in whom no deep-vein thrombosis developed. Both of these factors are associated with increased severity of injury or increased complications following injury, or both.
    Buerger PM; Peoples JB; Lemmon GW; and McCarthy MC: Risk of pulmonary emboli in patients with pelvic fractures. Am Surg,1993.59: 505-8, 59505  1993  [PubMed]
     
    Collins DN; Barnes CL; McCowan TC; Nelson CL; Carver DK; McAndrew MP; and Ferris EJ: Vena caval filter use in orthopaedic trauma patients with recognized preoperative venous thromboembolic disease. J Orthop Trauma,1992.6: 135-8, 6135  1992  [PubMed]
     
    Elliott CG; Dudney TM; Egger M; Orme JF; Clemmer TP; Horn SD; Weaver L; Handrahan D; Thomas F; Merrell S; Kitterman N; and Yeates S: Calf-thigh sequential pneumatic compression compared with plantar venous pneumatic compression to prevent deep-vein thrombosis after non-lower extremity trauma. J Trauma,1999.47: 25-32, 4725  1999  [PubMed]
     
    Geerts WH; Code KI; Jay RM; Chen E; and Szalai JP: A prospective study of venous thromboembolism after major trauma. N Engl J Med,1994.331: 1601-6, 3311601  1994  [PubMed]
     
    Knudson MM; Collins JA; Goodman SB; and McCrory DW: Thromboembolism following multiple trauma. J Trauma,1992.32: 2-11, 322  1992  [PubMed]
     
    Montgomery KD; Geerts WH; Potter HG; and Helfet DL: Thromboembolic complications in patients with pelvic trauma. Clin Orthop,1996.329: 68-87, 32968  1996  [PubMed]
     
    Webb LX; Rush PT; Fuller SB; and Meredith JW: Greenfield filter prophylaxis of pulmonary embolism in patients undergoing surgery for acetabular fracture. J Orthop Trauma,1992.6: 139-45, 6139  1992  [PubMed]
     
    Anglen JO; Goss K; Edwards J; and Huckfeldt RE: Foot pump prophylaxis for deep venous thrombosis: the rate of effective usage in trauma patients. Am J Orthop,1998.27: 580-2, 27580  1998  [PubMed]
     
    Fisher CG; Blachut PA; Salvian AJ; Meek RN; and O’Brien PJ: Effectiveness of pneumatic leg compression devices for the prevention of thromboembolic disease in orthopaedic trauma patients: a prospective, randomized study of compression alone versus no prophylaxis. J Orthop Trauma,1995.9: 1-7, 91  1995  [PubMed]
     
    Spain DA; Bergamini TM; Hoffmann JF; Carrillo EH; and Richardson JD: Comparison of sequential compression devices and foot pumps for prophylaxis of deep venous thrombosis in high-risk trauma patients. Am Surg,1998.64: 522-5, discussion 525-664522  1998  [PubMed]
     
    Decousus H; Leizorovicz A; Parent F; Page Y; Tardy B; Girard P; Laporte S; Faivre R; Charbonnier B; Barral FG; Huet Y; and Simonneau G: A clinical trial of vena caval filters in the prevention of pulmonary embolism in patients with proximal deep-vein thrombosis. Prevention du Risque d’Embolie Pulmonaire par Interruption Cave Study Group. N Engl J Med,1998.338: 409-15, 338409  1998  [PubMed]
     
    Fishmann AJ; Greeno RA; Brooks LR; and Matta JM: Prevention of deep vein thrombosis and pulmonary embolism in acetabular and pelvic fracture surgery. Clin Orthop,1994.305: 133-7, 305133  1994  [PubMed]
     
    Stover MDMorgan SJBosse MJSims SHHoward BJStackhouse DWeresh MJKellam JF. Prospective comparison of contrast enhanced computed tomography vs magnetic resonance venography in the detection of occult deep pelvic vein thrombosis in patients with pelvic and acetabular fractures. Read at the Annual Meeting of the Orthopaedic Trauma Association; 1998 Oct 8-10; Vancouver, British Columbia, Canada. 
     
    Ruedi TP, Murphy WM, editors. AO principles of fracture management. New York: Thieme; 2000. p 45-58. 
     
    Baker SP, O’Neill B, Haddon W Jr, Long WB: The injury severity score: a method for describing patients with multiple injuries and evaluating emergency care. J Trauma,1974.14: 187-96, 14187  1974  [PubMed]
     
    Knudson MM; Morabito D; Paiement GD; and Shackleford S: Use of low molecular weight heparin in preventing thromboembolism in trauma patients. J Trauma,1996.41: 446-59, 41446  1996  [PubMed]
     
    Kozak TK; Diebold R; and Beaver RJ: Massive pulmonary thromboembolism after manipulation of an unstable pelvic fracture: a case report and review of the literature. J Trauma,1995.38: 366-7, 38366  1995  [PubMed]
     
    Montgomery KD; Potter HG; and Helfet DL: Magnetic resonance venography to evaluate the deep venous system of the pelvis in patients who have an acetabular fracture. J Bone Joint Surg Am,1995.77: 1639-49, 771639  1995  [PubMed]
     
    Trottier SJ; Veremakis C; O’Brien J; and Auer AI: Femoral deep vein thrombosis associated with central venous catheterization: results from a prospective, randomized trial. Crit Care Med,1995.23: 52-9, 2352  1995  [PubMed]
     
    White RH; Goulet JA; Bray TJ; Daschbach MM; McGahan JP; and Hartling RP: Deep-vein thrombosis after fracture of the pelvis: assessment with serial duplex-ultrasound screening. J Bone Joint Surg Am,1990.72: 495-500, 72495  1990  [PubMed]
     
    Comerota AJ; Chouhan V; Harada RN; Sun L; Hosking J; Veermansunemi R; Comerota AJ Jr; Schlappy D; and Rao AK: The fibrinolytic effects of intermittent pneumatic compression: mechanism of enhanced fibrinolysis. Ann Surg,1997.226: 306-13, discussion 313-4226306  1997  [PubMed]
     

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    Buerger PM; Peoples JB; Lemmon GW; and McCarthy MC: Risk of pulmonary emboli in patients with pelvic fractures. Am Surg,1993.59: 505-8, 59505  1993  [PubMed]
     
    Collins DN; Barnes CL; McCowan TC; Nelson CL; Carver DK; McAndrew MP; and Ferris EJ: Vena caval filter use in orthopaedic trauma patients with recognized preoperative venous thromboembolic disease. J Orthop Trauma,1992.6: 135-8, 6135  1992  [PubMed]
     
    Elliott CG; Dudney TM; Egger M; Orme JF; Clemmer TP; Horn SD; Weaver L; Handrahan D; Thomas F; Merrell S; Kitterman N; and Yeates S: Calf-thigh sequential pneumatic compression compared with plantar venous pneumatic compression to prevent deep-vein thrombosis after non-lower extremity trauma. J Trauma,1999.47: 25-32, 4725  1999  [PubMed]
     
    Geerts WH; Code KI; Jay RM; Chen E; and Szalai JP: A prospective study of venous thromboembolism after major trauma. N Engl J Med,1994.331: 1601-6, 3311601  1994  [PubMed]
     
    Knudson MM; Collins JA; Goodman SB; and McCrory DW: Thromboembolism following multiple trauma. J Trauma,1992.32: 2-11, 322  1992  [PubMed]
     
    Montgomery KD; Geerts WH; Potter HG; and Helfet DL: Thromboembolic complications in patients with pelvic trauma. Clin Orthop,1996.329: 68-87, 32968  1996  [PubMed]
     
    Webb LX; Rush PT; Fuller SB; and Meredith JW: Greenfield filter prophylaxis of pulmonary embolism in patients undergoing surgery for acetabular fracture. J Orthop Trauma,1992.6: 139-45, 6139  1992  [PubMed]
     
    Anglen JO; Goss K; Edwards J; and Huckfeldt RE: Foot pump prophylaxis for deep venous thrombosis: the rate of effective usage in trauma patients. Am J Orthop,1998.27: 580-2, 27580  1998  [PubMed]
     
    Fisher CG; Blachut PA; Salvian AJ; Meek RN; and O’Brien PJ: Effectiveness of pneumatic leg compression devices for the prevention of thromboembolic disease in orthopaedic trauma patients: a prospective, randomized study of compression alone versus no prophylaxis. J Orthop Trauma,1995.9: 1-7, 91  1995  [PubMed]
     
    Spain DA; Bergamini TM; Hoffmann JF; Carrillo EH; and Richardson JD: Comparison of sequential compression devices and foot pumps for prophylaxis of deep venous thrombosis in high-risk trauma patients. Am Surg,1998.64: 522-5, discussion 525-664522  1998  [PubMed]
     
    Decousus H; Leizorovicz A; Parent F; Page Y; Tardy B; Girard P; Laporte S; Faivre R; Charbonnier B; Barral FG; Huet Y; and Simonneau G: A clinical trial of vena caval filters in the prevention of pulmonary embolism in patients with proximal deep-vein thrombosis. Prevention du Risque d’Embolie Pulmonaire par Interruption Cave Study Group. N Engl J Med,1998.338: 409-15, 338409  1998  [PubMed]
     
    Fishmann AJ; Greeno RA; Brooks LR; and Matta JM: Prevention of deep vein thrombosis and pulmonary embolism in acetabular and pelvic fracture surgery. Clin Orthop,1994.305: 133-7, 305133  1994  [PubMed]
     
    Stover MDMorgan SJBosse MJSims SHHoward BJStackhouse DWeresh MJKellam JF. Prospective comparison of contrast enhanced computed tomography vs magnetic resonance venography in the detection of occult deep pelvic vein thrombosis in patients with pelvic and acetabular fractures. Read at the Annual Meeting of the Orthopaedic Trauma Association; 1998 Oct 8-10; Vancouver, British Columbia, Canada. 
     
    Ruedi TP, Murphy WM, editors. AO principles of fracture management. New York: Thieme; 2000. p 45-58. 
     
    Baker SP, O’Neill B, Haddon W Jr, Long WB: The injury severity score: a method for describing patients with multiple injuries and evaluating emergency care. J Trauma,1974.14: 187-96, 14187  1974  [PubMed]
     
    Knudson MM; Morabito D; Paiement GD; and Shackleford S: Use of low molecular weight heparin in preventing thromboembolism in trauma patients. J Trauma,1996.41: 446-59, 41446  1996  [PubMed]
     
    Kozak TK; Diebold R; and Beaver RJ: Massive pulmonary thromboembolism after manipulation of an unstable pelvic fracture: a case report and review of the literature. J Trauma,1995.38: 366-7, 38366  1995  [PubMed]
     
    Montgomery KD; Potter HG; and Helfet DL: Magnetic resonance venography to evaluate the deep venous system of the pelvis in patients who have an acetabular fracture. J Bone Joint Surg Am,1995.77: 1639-49, 771639  1995  [PubMed]
     
    Trottier SJ; Veremakis C; O’Brien J; and Auer AI: Femoral deep vein thrombosis associated with central venous catheterization: results from a prospective, randomized trial. Crit Care Med,1995.23: 52-9, 2352  1995  [PubMed]
     
    White RH; Goulet JA; Bray TJ; Daschbach MM; McGahan JP; and Hartling RP: Deep-vein thrombosis after fracture of the pelvis: assessment with serial duplex-ultrasound screening. J Bone Joint Surg Am,1990.72: 495-500, 72495  1990  [PubMed]
     
    Comerota AJ; Chouhan V; Harada RN; Sun L; Hosking J; Veermansunemi R; Comerota AJ Jr; Schlappy D; and Rao AK: The fibrinolytic effects of intermittent pneumatic compression: mechanism of enhanced fibrinolysis. Ann Surg,1997.226: 306-13, discussion 313-4226306  1997  [PubMed]
     
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