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Effects of Limb-Length Discrepancy on Gait Economy and Lower-Extremity Muscle Activity in Older Adults
Burke Gurney, PhD, PT; Christine Mermier, MS; Robert Robergs, PhD; Anne Gibson, PhD; Dennis Rivero, MD
View Disclosures and Other Information
Investigation performed at the University of New Mexico, Albuquerque, New Mexico
Burke Gurney, PhD, PT
Division of Physical Therapy, University of New Mexico, HSSB 204B, Albuquerque, NM 87131-5661. E-mail address: bgurney@salud.unm.edu

Christine Mermier, MS
Robert Robergs, PhD
Anne Gibson, PhD
Division of Physical Performance Development, University of New Mexico, Johnson Center, Room B 143, Albuquerque, NM 87131-1251

Dennis Rivero, MD
Department of Orthopaedics, University of New Mexico, Health Sciences Center, Albuquerque, NM 87131-5296

No benefits in any form have been received or will be received from a commercial party related directly or indirectly to the subject of this article. Funds were received in total or partial support of the research or clinical study presented in this article. The funding source was an internal University of New Mexico Research Allocation Committee grant.

The Journal of Bone & Joint Surgery.  2001; 83:907-915 
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Abstract

Background: The amount of limb-length discrepancy necessary to adversely affect gait parameters in older adults is unknown, with information being largely anecdotal. This investigation was conducted to determine the effects of limb-length discrepancy on gait economy and lower-extremity muscle activity in older adults.

Methods: Forty-four men and women ranging in age from fifty-five to eighty-six years with no evidence of limb-length discrepancy of >1 cm participated in the study. Subjects walked on a treadmill at a self-selected normal walking pace with artificial limb-length discrepancies of 0, 2, 3, and 4 cm applied in a randomly selected order. Indirect calorimetry was used to measure oxygen consumption and minute ventilation. Electromyography was used to measure muscle activity of the right and left quadriceps femoris, plantar flexors, gluteus maximus, and gluteus medius. Heart rate, the rating of perceived exertion, and frequency of gait compensation patterns were also measured.

Results: There was a significant increase in oxygen consumption and the rating of perceived exertion with 2, 3, and 4-cm artificial limb-length discrepancies; a significant increase in heart rate, minute ventilation, and quadriceps activity in the longer limb with 3 and 4-cm artificial limb-length discrepancies; and a significant increase in plantar flexor activity in the shorter limb with a 4-cm artificial limb-length discrepancy compared with the same parameters with no artificial limb-length discrepancy.

Conclusions: Both oxygen consumption and the rating of perceived exertion were greater with a 2-cm artificial limb-length discrepancy than they were with no artificial limb-length discrepancy. There appears to be a breakpoint between 2 and 3 cm of artificial limb-length discrepancy in older adults with regard to the effects on most other physiological parameters. A 3-cm artificial limb-length discrepancy is likely to induce significant quadriceps fatigue in the longer limb. Elderly patients with substantial pulmonary, cardiac, or neuromuscular disease may have difficulty walking with a limb-length discrepancy as small as 2 cm.

Figures in this Article
    A measurable limb-length discrepancy is a relatively common problem. It is found in as many as 40%1 to 70%2 of the population, and a retrospective study demonstrated that a discrepancy of >2 cm affects at least one of every 1000 people3. The effects of limb-length discrepancy on function and the magnitude of limb-length discrepancy warranting treatment have been subjects of controversy for some time.
    Studies have been performed to investigate the effects of limb-length discrepancy on many functional parameters, including low-back pain4, osteoarthritis of the hip5, stress fractures6, aseptic loosening of hip prostheses7, standing balance8, forces transmitted through the hips9, running economy10, and associated running injuries11. Although no study has identified direct measures of gait economy with limb-length discrepancy, some indirect measures include ground-reaction forces, total mechanical work, and overall kinetic energy9,12-15. To our knowledge, Vink and Huson16 are the only authors who have used electromyography to examine the effects of limb-length discrepancy on muscle activity.
    On the basis of these studies, it appears that there is a breakpoint between 2 and 3 cm below which measurable changes do not take place. However, this information is largely anecdotal, and there has been disagreement about it17,18. Some of the disagreement may be due to the different populations (children and adults) used in studies of the effects of limb-length discrepancy19-21. Another factor might be the functional activities of the individual. For example, the amount of limb-length discrepancy that can cause symptoms in athletes may be substantially smaller than the amount that can cause symptoms in nonathletes22. Whereas Siffert23 reported that a limb-length discrepancy of 1.0 to 2.5 cm is rarely symptomatic in the general population, Friberg6 found that Finnish Army conscripts with as little as 1.0 cm of limb-length discrepancy had a greater incidence of stress fractures during extensive training than did those with no discrepancy. Subotnick1 proposed that 0.25 in (0.64 cm) of limb-length discrepancy in an athlete is as important pathologically as is 0.75 in (1.9 cm) in a nonathlete.
    Trauma, various diseases, and surgical procedures can result in a limb-length discrepancy. Perhaps the most common surgical procedure resulting in a limb-length discrepancy is total hip arthroplasty. More than 123,000 total hip arthroplasties were performed in the United States in 1994 alone24. The most common age of people undergoing total hip arthroplasty is fifty-five to eighty-five years25. Although there are a variety of complications associated with total hip arthroplasty, one of the most common is postoperative limb-length discrepancy26. The prevalence of limb-length discrepancy secondary to total hip arthroplasty has varied according to the study, from 27% with discrepancies large enough to require a shoe-lift27, to 18% with discrepancies of 1.5 cm28, to 16% with discrepancies of any magnitude29. If the more conservative figure of 16% were used, there would have been nearly 20,000 people with a measurable limb-length discrepancy secondary to total hip arthroplasty in 1994 in the United States alone.
    Investigators disagree regarding the magnitude of acceptable postoperative limb-length discrepancy. Some investigators have tried to quantify an important limb-length discrepancy, accepting as much as 2.0 cm, whereas others have defined an important discrepancy in terms of functional outcome30. The average limb-length discrepancy secondary to total hip arthroplasty has been reported to range from 3 to 19 mm31, with the actual values ranging from 0 to 3.0 cm28. Jasty et al.29 stated that, although limb-length discrepancies of 2 cm are common in the general population and may be asymptomatic, discrepancies after total hip arthroplasty lead to more symptoms. Surveys of patients have demonstrated that dissatisfaction after total hip arthroplasty is commonly due to limb-length discrepancy26. Edeen et al.31 interviewed patients postoperatively and found that, although their average limb-length discrepancy was only 0.97 cm, a substantial percentage (32%) were aware of the discrepancy. In addition, 24% of the patients, with an average limb-length discrepancy of 1.49 cm, overall were "annoyed" by it. Hozack et al.32 stated that limb-length discrepancy after total hip arthroplasty is one of the most common complaints in lawsuits against orthopaedic surgeons.
    Although investigators have studied the effects of limb-length discrepancy on many functional parameters, we are not aware of any study of how induced limb-length discrepancy affects older adults in terms of walking economy or muscle activity in the lower extremities. This information may help to quantify how much limb-length discrepancy can occur before functional gait is compromised. The purpose of this study was to investigate the effects of artificially induced limb-length discrepancies of 2, 3, and 4 cm on gait economy and lower-extremity muscle activity in older adults.
     
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    +Fig. 1:The response of oxygen consumption (VO2) to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
     
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    +Fig. 2:The response of the rating of perceived exertion (RPE) to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
     
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    +Fig. 3:The response of minute ventilation (VE) to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
     
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    +Fig. 4:The response of heart rate (HR) to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
     
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    +Fig. 5:The response of the integrated electromyographic (EMG) signal of the right quadriceps to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
     
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    +Fig. 6:The response of the integrated electromyographic (EMG) signal of the left plantar flexors to artificially induced limb-length discrepancy. The asterisk indicates a significant difference compared with the value with no discrepancy.
     
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    +Fig. 7:The overall average percent increases in all significant physiological variables in response to artificially induced limb-length discrepancy compared with the values with no artificial limb-length discrepancy.
     
    Anchor for JumpAnchor for JumpTABLE I:  Physical Characteristics and Demographic Data of the Sample of Forty-four Patients
    VariableRangeMean and Standard Deviation
    Age (yr)?55-86?71.8 ± 7.4
    Height (cm)?148.6-183.6165.3 ± 9.6
    Weight (kg)?48.7-99.5??72.0 ± 12.0
    Limb length (cm)75.00-96.6?86.5 ± 6.1
    Limb-length discrepancy (cm)?0.0-1.0??0.5 ± 0.3
    Walking pace on treadmill (mph)?1.10-3.00??1.6 ± 0.4
     
    Anchor for JumpAnchor for JumpTABLE II:  Summary of Significant Effects of Artificial Limb-Length Discrepancy
    Variable0-cm Discrepancy*2-cm Discrepancy*?3-cm ?Discrepancy*4-cm Discrepancy*
    Oxygen consumption (mL/kg/min)?9.26 ± 1.58?9.71 ± 1.57 ? 5%10.08 ± 1.65 ?9%10.49 ± 1.84 ?13%
    Rating of perceived exertion (6-20-point scale)?9.67 ± 2.1810.44 ± 2.07 ?8%10.75 ± 2.30 ?11%11.39 ± 2.30 ?18%
    Heart rate (bpm)89.58 ± 13.37??92.12 ± 14.34 ?3%?93.63 ± 13.78 ?5%
    Minute ventilation (L/min)22.69 ± 7.89?24.18 ± 7.11 ?7%25.47 ± 8.43 ?12%
    R quadriceps activity (V)13.29 ± 10.37??20.50 ± 17.46 ?54%?22.72 ± 16.63 ?71%
    L plantar flexor activity (V)32.17 ± 25.10???45.31 ± 36.31 ?41%
    *The values are given as the average and the standard deviation. ? = no significant change, with the numbers available, and = an increase from the value with no artificial limb-length discrepancy.

    Setting

    All physiological testing was conducted from August to December 1999 at the Human Performance Laboratory at the University of New Mexico in Albuquerque, New Mexico. The same well-lit, temperature-controlled room was used for all testing. All data were collected from each subject during one test session.

    Subjects

    Forty-four patients (nineteen men and twenty-five women) ranging in age from fifty-five to eighty-six years were recruited as a sample of convenience (Table I). They were screened for limb-length discrepancy with use of a standard flexible tape measure, with each limb measured from the medial malleolus to the anterior inferior iliac spine as recommended by Beattie et al.33. Individuals who had limb lengths within 1 cm of each other and who did not have a neurological, orthopaedic, severe pulmonary, or cardiac disorder or a history of falls were included in the study. After being accepted as participants in the study, the subjects were randomly assigned an order for the application of the shoe-lifts (the artificial limb-length discrepancies).
    This study was approved by the School of Medicine Human Research Review Committee at the University of New Mexico. Prior to testing, all subjects received explanations of the testing procedures and provided written informed consent.

    Measures

    Heart rate: The subject’s heart rate was measured with a heart rate telemetry unit (Polar Heart Rate Monitor, model 1901201; Polar Electro, Woodbury, New York). Heart rates were recorded at rest, at a steady state of each condition, and during rest between each trial to establish adequate rest.
    Rating of perceived exertion: Subjects received verbal instruction regarding subjective scoring of perceived exertion with use of Borg’s original rating scale of 6 to 20 points34.
    Muscle activity: The subject’s skin was prepared for the surface electromyographic electrodes by rubbing for fifteen seconds with an alcohol preparation. Blue Sensor surface electrodes (Medicotest, Rugmarken, Denmark) were applied parallel to the muscle fibers on the muscle belly. In all cases, the electrode placement was checked by asking the patient to contract the respective muscle as well as other muscle groups in the region and inspecting the electromyographic readout for cross talk. The electrodes were connected to a Noraxon Myosystem-1200 sixteen-channel electromyographic machine (Noraxon USA, Scottsdale, Arizona). Each rectified burst of electromyographic activity had a trigger level of 2 V, an onset time of twenty milliseconds, and a subsist time of twenty milliseconds. Bursts of the electromyographic signal were then filtered with a Butterworth low-pass filter at 250 Hz and were integrated with use of Myosoft (Noraxon USA) software. A per-burst average was calculated from a fifteen-second trial and was recorded in microvolts as the integrated electromyographic signal. This measure was used to depict muscle activity. The muscle activities of the right and left ankle plantar flexors, quadriceps femoris, gluteus maximus, and gluteus medius were recorded throughout all trials.
    Oxygen consumption and minute ventilation: Expired gas was collected every thirty seconds with use of a Jaeger ergo-oxyscreen computerized expired-gas-analysis system (Erich Jaeger, Wurtzburg, Germany) during the submaximal treadmill tests (Trackmaster Treadmill; JAS Fitness Systems, Carrolton, Texas) at a self-selected normal walking pace as subjects breathed through a T-configuration, one-way valved mouthpiece (Hans Rudolph, Kansas City, Missouri). When the values of absolute oxygen consumption remained within 75 mL O2/min for a minimum of three consecutive thirty-second readings (the steady state), the values were recorded as the submaximal oxygen consumption and the minute ventilation, and the subject was told to stop walking. The error of the pneumotach is 1%, and the analyzer had no measurable error; therefore, the sensitivity of the measurements of minute ventilation and oxygen consumption was 1%.

    Shoe-Lifts

    All shoes used by an individual subject were of the same make, model, and size. A 2 or 4-cm shoe-lift constructed from crepe material was glued into the midsole of the right shoe, and then the outsole of the shoe was replaced. To attain 3 cm of artificial limb-length discrepancy, a 1-cm insole insert was added to the 2-cm midsole lift.

    Physical Activity

    Subjects were screened for their level of physical activity with use of the modified Baecke questionnaire for elderly individuals35.

    Testing

    Subjects walked on the treadmill, without use of the handrails, starting at 1 mph. The speed of the treadmill was increased at a rate of 0.5 mph every fifteen seconds until the subjects reported their self-selected normal walking pace. Subjects who were unfamiliar with the use of a treadmill were allowed to practice until they felt comfortable walking on the treadmill and the investigator determined that their gait appeared normal. Subjects then walked on the treadmill at their self-selected normal walking pace with different amounts of artificial limb-length discrepancy applied in a randomly assigned order. The subject’s oxygen consumption and minute ventilation were measured after a steady state was achieved at each level. Electromyographic data, heart rate, and rating of perceived exertion were recorded throughout a fifteen-second trial after a steady state was attained. The average duration of each run was slightly more than six minutes. To prevent fatigue, subjects rested between trials until their heart rate was within 5 bpm of their resting heart rate.

    Statistical Analysis

    A repeated-measures analysis of variance was used to assess the overall effect of artificial limb-length discrepancy on oxygen consumption, minute ventilation, rating of perceived exertion, heart rate, and activity of the right and left ankle plantar flexors, quadriceps femoris, gluteus maximus, and gluteus medius during gait. Tukey post hoc tests were then used on significant findings to compare gait with no artificial limb-length discrepancy with gait with 2, 3, and 4 cm of artificial limb-length discrepancy for the above parameters. In addition, the subject’s age, gender, height, limb length, treadmill experience, and physical activity were analyzed with use of repeated-measures analysis of variance to determine whether they played a role in the effect of artificial limb-length discrepancy on the above parameters. Significance was set at p £ 0.05. Effect size was calculated for all significant findings.

    Oxygen Consumption

    Artificially induced limb-length discrepancy had a significant overall effect on oxygen consumption (p < 0.0005, effect size = 0.56). There was a significant increase in oxygen consumption when subjects walked with a 4-cm discrepancy (mean, 10.49 mL O2/kg/min; p < 0.0005), a 3-cm discrepancy (mean, 10.08 mL O2/kg/min; p < 0.0005), and a 2-cm discrepancy (mean, 9.71 mL O2/kg/min; p < 0.0005) compared with when they walked with no discrepancy (mean, 9.26 mL O2/kg/min) (Fig. 1; Table II).

    Rating of Perceived Exertion

    Artificial limb-length discrepancy also had a significant overall effect on the rating of perceived exertion (p < 0.0005, effect size = 0.449). Subjects reported a higher rating of perceived exertion when they walked with a 4-cm discrepancy (mean, 11.39; p < 0.0005), a 3-cm discrepancy (mean, 10.75; p < 0.0005), and a 2-cm discrepancy (mean, 10.44; p = 0.013) compared with when they walked with no discrepancy (mean, 9.67) (Fig. 2; Table II).

    Minute Ventilation

    Minute ventilation was significantly affected by artificial limb-length discrepancy as well (p < 0.0005, effect size = 0.524). Subjects had increased minute ventilation when they walked with a 4-cm discrepancy (mean, 25.47 L/min; p < 0.0005) and a 3-cm discrepancy (mean, 24.18 L/min; p = 0.013) than when they walked with no discrepancy (mean, 22.69 L/min) (Fig. 3; Table II).

    Heart Rate

    Artificial limb-length discrepancy had a significant overall effect on heart rate (p < 0.0005, effect size = 0.459). Subjects had a higher heart rate when they walked with a 4-cm discrepancy (mean, 93.63 bpm; p < 0.0005) and a 3-cm discrepancy (mean, 92.12 bpm; p = 0.001) than when they walked with no discrepancy (mean, 89.58 bpm) (Fig. 4; Table II).

    Integrated Electromyographic Signal

    Right quadriceps femoris muscle: There was a significant overall effect of artificial limb-length discrepancy on the activity of the right quadriceps femoris muscle (p < 0.0005, effect size = 0.392). Subjects had higher activity when they walked with a 4-cm discrepancy (mean, 22.72 V; p < 0.0005) and a 3-cm discrepancy (mean, 20.50 V; p = 0.001) than when they walked with no discrepancy (mean, 13.29 V) (Fig. 5; Table II).
    Left plantar flexor muscles: Artificial limb-length discrepancy also had a significant overall effect on the activity of the left plantar flexors (p = 0.042, effect size = 0.246). When subjects walked with a 4-cm discrepancy, they had higher activity (mean, 45.31 V) than when they walked with no discrepancy (mean, 32.17 V; p < 0.003) (Fig. 6; Table II).

    Effects of Age, Gender, Height, Limb Length, Treadmill Experience, and Physical Activity

    With the numbers available, age, gender, height, limb length, treadmill experience, and physical activity had no significant effect on any of the above parameters.

    Qualitative Observations of Gait with Artificial Limb-Length Discrepancy

    Observations of gait with the artificial limb-length discrepancies revealed that the most common primary compensatory strategy was steppage gait (increased hip and knee flexion), which was used by twenty-one of the forty-four subjects. This was followed in frequency by circumduction (increased hip abduction at swing phase), used by eight subjects; vaulting (increased plantar flexion at step phase), used by five; and hip-hiking (increased ipsilateral lumbar side flexion at swing phase), used by two. Subjects often used more than one, and as many as three, compensatory strategies simultaneously while walking on the treadmill. A total of ten distinct gait-compensation strategies were observed. Eight subjects did not demonstrate a consistent observable gait compensation.

    Problems Experienced by Subjects When Walking with Artificial Limb-Length Discrepancy

    On the whole, subjects had few problems when walking with an artificial limb-length discrepancy. Several stated that they felt as if they were walking on the side of a hill or with the right foot up on a curb. One subject who had reported having arthritis in the right knee complained of pain in that knee with the 4-cm artificial limb-length discrepancy but insisted on completing the trial. Another subject reported low-back pain when walking with both the 3 and 4-cm artificial limb-length discrepancy but also insisted on completing both trials. Three subjects dragged the right foot when wearing the 3 and 4-cm lifts, and two subjects lost their balance several times when walking with the 4-cm limb-length discrepancy and had to momentarily grab the handrail.
    A large number of studies have been performed in an attempt to determine the magnitude of limb-length discrepancy necessary to manifest complications and, therefore, to warrant treatment3,6,19,22,23,36. However, controversy still remains regarding the breakpoint between acceptable and unacceptable discrepancy, especially in older adults.
    The significant increases in oxygen consumption, minute ventilation, heart rate, and rating of perceived exertion in this study were probably a result, at least in part, of an increase in the subjects’ vertical displacement of the center of mass37. When a subject is in the stance phase of the longer limb, there is a tendency to raise the center of mass of the head, abdomen, and trunk. In fact, this increase in vertical displacement of the head, abdomen, and trunk has been shown to be a strong predictor (r2 = 0.91) of oxygen consumption by normal subjects walking at variable speeds38. The increases seen in quadriceps activity with 3 and 4 cm of artificial limb-length discrepancy are consistent with the increases in ground-reaction force of the longer limb reported by several authors12,14. The increased ground-reaction force places a greater demand on the quadriceps at heel-strike. It is probable that the subjects’ attempts to reduce the length of the right (longer) limb by maintaining it in slight flexion during stance phase could also account for the increase in the output of the right quadriceps. The overall increase seen in the activity of the left plantar flexors with the 4-cm artificial limb-length discrepancy could be due to the ten subjects who demonstrated a vaulting gait (five of whom used vaulting as the primary compensatory strategy and five of whom used it as a secondary compensatory strategy). These plantar flexors would be firing during the swing phase of the right limb to gain additional clearance of the longer limb.
    Although no study has identified direct measures of gait economy with limb-length discrepancies, some indirect measurements have been performed. Bhave et al.12 found that limb-length discrepancy creates an asymmetry in ground-reaction force and that surgical lengthening of the short limb to within 1 cm of the other limb reduced the asymmetry to below significance. Kaufman et al.14 found that a discrepancy of more than 2 cm was necessary to result in asymmetry of the ground-reaction force. Liu et al.15 also measured ground-reaction force and concluded that gait asymmetries were not evident until a limb-length discrepancy was in excess of 2.33 cm. Brand and Yack9 found that a 2.3-cm artificial limb-length discrepancy did not change ground-reaction force at the hip but a 3.5-cm discrepancy did. Delacerda and Wikoff13 found that use of a corrective shoe by a woman with a 2.87-cm limb-length discrepancy significantly reduced the kinetic energy expended during walking (p < 0.05). We know of only one study in which electromyography was used to measure the effects of limb-length discrepancy on gait. Vink and Huson16 found that there was a significant increase in the electromyographic activity of the erector spinae only when the limb-length discrepancy was 3 cm. On the basis of these studies, it appears that a limb-length discrepancy of slightly >2 cm is a breakpoint above which there are measurable changes in gait. In fact, it is generally accepted that a 2-cm limb-length discrepancy represents the breakpoint between treatment and no treatment. However, this information is largely anecdotal and there has been disagreement about it. For example, Song et al.21 found that asymmetries in mechanical work performed by the long extremity were observable when the limb-length discrepancy was as small as 1.64 cm.
    In our study, overall increases in physiological parameters were not equal between successive incremental increases in artificial limb-length discrepancy. Figure 7 shows the overall average percent increases in all significant physiological variables compared with the values associated with no artificial limb-length discrepancy. The 5.6% overall increase in physiological parameters between 0 and 2 cm of artificial limb-length discrepancy was small when compared with the 13.3% increase between 0 and 3 cm and the 24.1% increase between 0 and 4 cm. It is noteworthy that the percent increase in physiological parameters between 0 and 2 cm of artificial limb-length discrepancy averaged 2.8%/cm, whereas it averaged 7.7% between 2 and 3 cm and 10.8% between 3 and 4 cm of artificial limb-length discrepancy. These findings are in agreement with those of several authors who reported that changes in gait did not take place until limb-length discrepancy was in excess of 2 cm2,12-15. These findings should be interpreted with caution, however, since in our study oxygen consumption and, perhaps most importantly, the rating of perceived exertion were significantly increased with 2 cm of artificial limb-length discrepancy (p < 0.0005 and p = 0.013, respectively). We found that most increases seen with 4 cm of artificial limb-length discrepancy were large (average increase, 24%), which is contrary to the findings of Phelps et al.17, who reported that alterations in oxygen consumption outside the range of normal were not seen in young athletes even with limb-length discrepancies of 6 cm, and also to those of Richter18, who observed no changes in heart rate until his subjects had 4 cm of artificial limb-length discrepancy. The fact that our study dealt with older adults could account for the difference between our findings and those of Phelps et al., who performed their study on young adults. In Richter’s study, the only outcome was heart rate, which does not "tell the whole story" because increases in energy expenditure can be absorbed by increases in stroke volume and/or peripheral extraction as well as heart rate.
    According to Waters et al.39, unlimited endurance requires that the energy cost for walking be <50% maximum oxygen consumption. It is at about 50% maximum oxygen consumption that individuals who are not physically fit experience lactate threshold36. Therefore, from an oxygen-consumption perspective, an otherwise healthy individual would be able to walk with a 4-cm limb-length discrepancy indefinitely. The same would be the case for minute ventilation. In our study, the minute ventilation seen with a 4-cm artificial limb-length discrepancy was 25.47 L/min, which is 24% of the maximum minute ventilation of a typical seventy to seventy-nine-year-old man40. The average maximum oxygen consumption of older people (sixty-five years and older) with a poor fitness status is about 25 mL O2/kg/min41. The average oxygen consumption of our subjects walking with a 4-cm artificial limb-length discrepancy was 10.49 mL O2/kg/min, or 42% of maximum oxygen consumption.
    If an older person has compromised cardiac and/or pulmonary status, however, a limb-length discrepancy as small as 2 cm could pose a problem. The maximum oxygen consumption of a person with severe congestive heart failure can be as low as 10 to 15 mL O2/kg/min42,43. In fact, Meyer et al.44 found that, on the average, the lactate threshold occurred at 9.3 ± 0.4 mL O2/kg/min in eighteen patients with severe congestive heart failure. In our study, a 2-cm artificial limb-length discrepancy raised oxygen consumption from 9.26 to 9.71 mL O2/kg/min. If patients with congestive heart failure had a 2-cm limb-length discrepancy, they would have to have a maximum oxygen consumption of at least 19.42 mL O2/kg/min (50% maximum oxygen consumption) to walk for a prolonged period of time. The maximum oxygen consumption would have to be 20.16 mL O2/kg/min if the limb-length discrepancy were 3 cm and 20.98 mL O2/kg/min if the limb-length discrepancy were 4 cm. The average oxygen consumption expenditures were measured at an average walking pace of only 1.6 mph in this study.
    Similarly, patients with severe chronic obstructive pulmonary disease or restrictive lung disease can have a maximum minute ventilation as low as 25 to 30 L/min45. These individuals would almost reach their maximum minute ventilation (97% to 81%) if they had a 3-cm limb-length discrepancy, and they would be affected by even this moderate increase in minute ventilation. With a 4-cm limb-length discrepancy, patients with a maximum minute ventilation of 25 L/min would exceed their maximum value whereas individuals with a maximum minute ventilation as high as 32 L/min would be at 80% of their maximum minute ventilation.
    The 54% increase in the activity of the right quadriceps muscle seen with the 3-cm artificial limb-length discrepancy could be a problem for patients with compromised limb strength, as might result from a stroke. Patients who have had a stroke are more likely to fall onto their affected side (chi square = 22.5, p < 0.001)46, and a patient who has done this and has sustained a subcapital hip fracture necessitating a total hip arthroplasty would be especially susceptible to the problems created by a limb-length discrepancy after surgery.
    It is generally accepted that overfatigue of the muscles of people with multiple sclerosis47, Guillain-Barré syndrome48, and postpoliomyelitis syndrome49 can be detrimental. Since individuals with these disorders have substantially compromised endurance with repeated high-intensity contractions, a limb-length discrepancy could limit the distance that these individuals are able to travel before fatigue. The 41% increase in the activity of the left plantar flexors and the 71% increase in that of the right quadriceps muscle seen with the 4-cm artificial limb-length discrepancy in our study could be clinically relevant.

    Gait Compensation Strategies

    The fact that artificial limb-length discrepancy of as much as 4 cm resulted in no significant increases in the activity of any of the other muscle groups deserves some consideration. At least four distinct compensation strategies (steppage, circumduction, vaulting, and hip-hiking) emerged during gait, with some subjects using more than one, and as many as three, strategies simultaneously. In all, ten different combinations of strategies were apparent. In addition, three subjects were observed dragging the right foot repeatedly, and two subjects lost their balance several times. The result of this variety of strategies would be an equal variety of muscle recruitment patterns. Therefore, no one muscle group had greater activity in more than a few individuals. This probably explains the lack of significant increases in the activities of the other muscle groups.

    Limitations of the Study

    This study addressed the acute effects of limb-length discrepancy. Chronic adaptations are possible and may reduce the amount of functional loss over time. An example of an adaptation is pelvic obliquity, which is an efficient way to reduce increases in vertical center-of-mass oscillations. However, in an older adult, this compensation may not be possible because of stiffness in the spine and decreased spinal motion. In addition, the shoe-lifts added an average weight of 72 g to the shoe. This added weight might have been responsible for some of the increases in the physiological parameters. Even though subjects were allowed as long as twenty minutes to adapt to treadmill walking, thirteen of the subjects had limited experience with walking on a treadmill and, for them, it may not have been representative of walking on the ground.

    Conclusions

    An artificial limb-length discrepancy of 2 cm had a significant effect on oxygen consumption and the rating of perceived exertion. Older adults appear to have a breakpoint between 2 and 3 cm of artificial limb-length discrepancy that determines the effect on most other physiological parameters, although 3 cm appears to be the breakpoint for significant quadriceps fatigue in the longer limb as it causes a 54% increase in quadriceps activity. Elderly patients with substantial compromise of pulmonary, cardiac, or neuromuscular function may have difficulty walking with a limb-length discrepancy as small as 2 cm.
    It would be instructive to repeat this study with the use of a portable gas-analysis system and to expand it to activities of daily living.
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    Gofton JP, and Trueman GE: Studies in osteoarthritis of the hip. II. Osteoarthritis of the hip and leg-length disparity. Can Med Assoc J,1971.104: 791-9, 104791  1971  [PubMed]
     
    Friberg O: Leg length asymmetry in stress fractures. A clinical and radiological study. J Sports Med Phys Fitness,1982.22: 485-8, 22485  1982  [PubMed]
     
    Visuri T; Lindholm TS; Antti-Poika I; and Koskenvou M: The role of overlength of the leg in aseptic loosening after total hip arthroplasty. Ital J Orthop Traumatol,1993.19: 107-11, 19107  1993  [PubMed]
     
    Murrell P; Cornwall MW; and Doucet SK: Leg-length discrepancy: effect on the amplitude of postural sway. Arch Phys Med Rehabil,1991.72: 646-8, 72646  1991  [PubMed]
     
    Brand RA, and Yack HJ: Effects of leg length discrepancies on the forces at the hip joint. Clin Orthop,1996.333: 172-80, 333172  1996  [PubMed]
     
    Delacerda FG, and McCrory ML: A case report: effect of a leg length differential on oxygen consumption. J Orthop Sports Phys Ther,1981.3: 17-20, 317  1981  [PubMed]
     
    Brunet ME; Cook SD; Brinker MR; and Dickinson JA: A survey of running injuries in 1505 competitive and recreational runners. J Sports Med Phys Fitness,1990.30: 307-15, 30307  1990  [PubMed]
     
    Bhave A; Paley D; and Herzenberg JE: Improvement in gait parameters after lengthening for the treatment of limb-length discrepancy. J Bone Joint Surg Am,1999.81: 529-34, 81529  1999  [PubMed]
     
    Delacerda FG, and Wikoff OD: Effect of lower extremity asymmetry on the kinematics of gait. J Orthop Sports Phys Ther,1982.3: 105-7, 3105  1982  [PubMed]
     
    Kaufman KR; Miller LS; and Sutherland DH: Gait asymmetry in patients with limb-length inequality. J Pediatr Orthop,1996.16: 144-50, 16144  1996  [PubMed]
     
    Liu XC; Fabry G; Molenaers G; Lammens J; and Moens P: Kinematic and kinetic asymmetry in patients with leg-length discrepancy. J Pediatr Orthop,1998.18: 187-9, 18187  1998  [PubMed]
     
    Vink P, and Huson A: Lumbar back muscle activity during walking with a leg inequality. Acta Morphol Neerl Scand,1987.25: 261-71, 25261  1987  [PubMed]
     
    Phelps JA, Novacheck TA, Dahl MT. Consequences of leg length inequality in young adults. Read at the Annual East Coast Clinical Gait Laboratory Conference; 1993 May 6; Rochester, MN. 
     
    Richter J: [Differences in the length of legs and walking capacity (a contributionto ergometry in orthopedics)]. Z Orthop Ihre Grenzgeb,1968.104: 548—54, German104548  1968 
     
    Dahl MT: Limb length discrepancy. Pediatr Clin North Am,1996.43: 849—65, 43849  1996  [PubMed]
     
    Etnier JL, and Landers DM: Motor performance and motor learning as a function of age andfitness. Res Q Exerc Sport,1998.69: 136—46, 69136  1998  [PubMed]
     
    Song KM; Halliday SE; and Little DG: The effect of limb-length discrepancy on gait. J Bone Joint Surg Am,1997.79: 1690—8, 791690  1997  [PubMed]
     
    Subotnick SI: The short leg syndrome. J Am Podiatry Assoc,1976.66: 720—3, 66720  1976  [PubMed]
     
    Siffert RS:J Bone Joint Surg Am,1987.69: 1100—6, 691100  1987  [PubMed]
     
    Towheed TE, and Hochberg MC: Health-related quality of life after total hip replacement. Semin Arthritis Rheum,1996.26: 483-91, 26483  1996  [PubMed]
     
    Cauley JA. Epidemiology of total hip replacement. NIH Consensus Development Conference on Total Hip Replacement. Symposium conducted at the National Institutes of Health Annual Conference; 1994 Sept 12; Bethesda, MD. 
     
    O’Brien S; Engela DW; Trainor P; and Beverland DE: Assessing the accuracy of femoral component placement in custom cemented hip replacement. Orthop Nurs. ,1996.15: 47-53, 1547  1996  [PubMed]
     
    Williamson JA, and Reckling FW: Limb length discrepancy and related problems following total hip replacement. Clin Orthop,1978.134: 135-8, 134135  1978  [PubMed]
     
    Love BRT, and Wright K: Leg length discrepancy after total hip joint replacement. In: Proceedings of the Australian Orthopaedic Association. J Bone Joint Surg Br. ,1983.65: 103, 65103  1983 
     
    Jasty M; Webster W; and Harris W: Management of limb length inequality during total hip replacement. Clin Orthop,1996.333: 165-71, 333165  1996  [PubMed]
     
    Abraham WD: Leg length discrepancy in total hip arthroplasty. Orthop Clin North Am,1992.23: 201-9, 23201  1992  [PubMed]
     
    Edeen J; Sharkey PF; and Alexander AH: Clinical significance of leg-length inequality after total hip arthroplasty. Am J Orthop.,1995.24: 347-51, 24347  1995  [PubMed]
     
    Hozack WJ, Lavernia CJ, Ranawat CS. Achieving stability and leg length equality in total hip arthroplasty, acetabular considerations. Instructional Course Lecture at the Annual Meeting of the American Academy of Orthopaedic Surgeons; 2000 March 15-19; Orlando, FL. 
     
    Beattie P; Isaacson K; Riddle DL; and Rothstein JM: Validity of derived measurements of leg-length differences obtained by use of a tape measure. Phys Ther. ,1990.70: 150-7, 70150  1990  [PubMed]
     
    Borg G: Psychophysical bases of perceived exertion. Med Sci Sports Exerc. ,1982.14: 377-81, 14377  1982  [PubMed]
     
    Voorrips LE; Ravelli AC; Dongelmans PC; Deurenberg P; and Van Staveren WA: A physical activity questionnaire for the elderly. Med Sci Sports Exerc,1991.23: 974-9, 23974  1991  [PubMed]
     
    Wilmore JH, Costill DL. Physiology of sport and exercise. 3rd ed. Champaign, IL: Human Kinetics; 1999. p 139. 
     
    Perry J. Gait analysis: normal and pathological function. Thorofare, NJ: Slack; 1992. p 40-1. 
     
    Waters RL, and Mulroy S: Energy expenditure of normal and pathologic gait. Gait Posture,1999.9: 207-31, 9207  1999  [PubMed]
     
    Waters RL; Hislop HJ; Perry J; and Antonelli D: Energetics: application to the study and management of locomotor disabilities. Energy cost of normal and pathologic gait. Orthop Clin North Am,1978.9: 351-6, 9351  1978  [PubMed]
     
    Cander L, Moyer JH. Aging and the lung. Perspectives. New York: Grune and Stratton; 1964. p 132. 
     
    Heyward VH. Advanced fitness assessment and exercise prescription. 2nd ed. Champaign, IL: Human Kinetics; 1991. p 48. 
     
    Lucas C; Stevenson LW; Johnson W; Hartley H; Hamilton MA; Walden J; Lem V; and Eagen-Bengsten E: The 6-min walk and peak oxygen consumption in advanced heart failure: aerobic capacity and survival. Am Heart J,1999.138: 618-24, 138618  1999  [PubMed]
     
    Poole-Wilson PA, Colucci WS, Massie BM, Chatterjee K, Coats AJS. Heart failure: scientific principles and clinical practices. New York: Churchill Livingstone; 1997. p 447. 
     
    Meyer K; Gornandt L; Schwaibold M; Westbrook S; Hajric R; Peters K; Beneke R; Schnellbacher K; and Roskamm H: Predictors of response to exercise training in severe chronic congestive heart failure. Am J Cardiol,1997.80: 56-60, 8056  1997  [PubMed]
     
    Jones NL; Jones G; and Edwards RH: Exercise tolerance in chronic airway obstruction. Am Rev Respir Dis,1971.103: 477-91, 103477  1971  [PubMed]
     
    Ramnemark A; Nyberg L; Borssen B; Olsson T; and Gustafson Y.: Fractures after stroke. Osteoporos Int,1998.8: 92-5, 892  1998  [PubMed]
     
    Costello E; Curtis CL; Sandel IB; and Bassile CC: Exercise prescription for individuals with multiple sclerosis. Neurol Rep,1996.20: 24-30, 2024  1996 
     
    Bassille CC: Guillain-Barré syndrome and exercise guidelines. Neurol Rep,1996.20: 31-6, 2031  1996 
     
    McDonald-Williams MF: Exercise and postpolio syndrome. Neurol Rep,1996.20: 37-44, 2037  1996 
     

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    Anchor for JumpAnchor for Jump
    +Fig. 1:The response of oxygen consumption (VO2) to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
    Anchor for JumpAnchor for Jump
    +Fig. 2:The response of the rating of perceived exertion (RPE) to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
    Anchor for JumpAnchor for Jump
    +Fig. 3:The response of minute ventilation (VE) to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
    Anchor for JumpAnchor for Jump
    +Fig. 4:The response of heart rate (HR) to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
    Anchor for JumpAnchor for Jump
    +Fig. 5:The response of the integrated electromyographic (EMG) signal of the right quadriceps to artificially induced limb-length discrepancy. The asterisks indicate a significant difference compared with the value with no discrepancy.
    Anchor for JumpAnchor for Jump
    +Fig. 6:The response of the integrated electromyographic (EMG) signal of the left plantar flexors to artificially induced limb-length discrepancy. The asterisk indicates a significant difference compared with the value with no discrepancy.
    Anchor for JumpAnchor for Jump
    +Fig. 7:The overall average percent increases in all significant physiological variables in response to artificially induced limb-length discrepancy compared with the values with no artificial limb-length discrepancy.
    Anchor for JumpAnchor for JumpTABLE I:  Physical Characteristics and Demographic Data of the Sample of Forty-four Patients
    VariableRangeMean and Standard Deviation
    Age (yr)?55-86?71.8 ± 7.4
    Height (cm)?148.6-183.6165.3 ± 9.6
    Weight (kg)?48.7-99.5??72.0 ± 12.0
    Limb length (cm)75.00-96.6?86.5 ± 6.1
    Limb-length discrepancy (cm)?0.0-1.0??0.5 ± 0.3
    Walking pace on treadmill (mph)?1.10-3.00??1.6 ± 0.4
    Anchor for JumpAnchor for JumpTABLE II:  Summary of Significant Effects of Artificial Limb-Length Discrepancy
    Variable0-cm Discrepancy*2-cm Discrepancy*?3-cm ?Discrepancy*4-cm Discrepancy*
    Oxygen consumption (mL/kg/min)?9.26 ± 1.58?9.71 ± 1.57 ? 5%10.08 ± 1.65 ?9%10.49 ± 1.84 ?13%
    Rating of perceived exertion (6-20-point scale)?9.67 ± 2.1810.44 ± 2.07 ?8%10.75 ± 2.30 ?11%11.39 ± 2.30 ?18%
    Heart rate (bpm)89.58 ± 13.37??92.12 ± 14.34 ?3%?93.63 ± 13.78 ?5%
    Minute ventilation (L/min)22.69 ± 7.89?24.18 ± 7.11 ?7%25.47 ± 8.43 ?12%
    R quadriceps activity (V)13.29 ± 10.37??20.50 ± 17.46 ?54%?22.72 ± 16.63 ?71%
    L plantar flexor activity (V)32.17 ± 25.10???45.31 ± 36.31 ?41%
    *The values are given as the average and the standard deviation. ? = no significant change, with the numbers available, and = an increase from the value with no artificial limb-length discrepancy.
    Subotnick SI: Limb length discrepancies of the lower extremity (the short leg syndrome). J Orthop Sports Phys Ther,1981.3: 11-5, 311  1981  [PubMed]
     
    Woerman AL, and Binder-MacLeod SA: Leg length discrepancy assessment: accuracy and precision in five clinical methods of evaluation. J Orthop Sports Phys Ther,1984.5: 230-8, 5230  1984  [PubMed]
     
    Guichet JM; Spivak JM; Trouilloud P; and Grammont PM: Lower limb-length discrepancy. An epidemiologic study. Clin Orthop,1991.272: 235-41, 272235  1991  [PubMed]
     
    Giles LG, and Taylor JR: Low-back pain associated with leg length inequality. Spine,1981.6: 510-21, 6510  1981  [PubMed]
     
    Gofton JP, and Trueman GE: Studies in osteoarthritis of the hip. II. Osteoarthritis of the hip and leg-length disparity. Can Med Assoc J,1971.104: 791-9, 104791  1971  [PubMed]
     
    Friberg O: Leg length asymmetry in stress fractures. A clinical and radiological study. J Sports Med Phys Fitness,1982.22: 485-8, 22485  1982  [PubMed]
     
    Visuri T; Lindholm TS; Antti-Poika I; and Koskenvou M: The role of overlength of the leg in aseptic loosening after total hip arthroplasty. Ital J Orthop Traumatol,1993.19: 107-11, 19107  1993  [PubMed]
     
    Murrell P; Cornwall MW; and Doucet SK: Leg-length discrepancy: effect on the amplitude of postural sway. Arch Phys Med Rehabil,1991.72: 646-8, 72646  1991  [PubMed]
     
    Brand RA, and Yack HJ: Effects of leg length discrepancies on the forces at the hip joint. Clin Orthop,1996.333: 172-80, 333172  1996  [PubMed]
     
    Delacerda FG, and McCrory ML: A case report: effect of a leg length differential on oxygen consumption. J Orthop Sports Phys Ther,1981.3: 17-20, 317  1981  [PubMed]
     
    Brunet ME; Cook SD; Brinker MR; and Dickinson JA: A survey of running injuries in 1505 competitive and recreational runners. J Sports Med Phys Fitness,1990.30: 307-15, 30307  1990  [PubMed]
     
    Bhave A; Paley D; and Herzenberg JE: Improvement in gait parameters after lengthening for the treatment of limb-length discrepancy. J Bone Joint Surg Am,1999.81: 529-34, 81529  1999  [PubMed]
     
    Delacerda FG, and Wikoff OD: Effect of lower extremity asymmetry on the kinematics of gait. J Orthop Sports Phys Ther,1982.3: 105-7, 3105  1982  [PubMed]
     
    Kaufman KR; Miller LS; and Sutherland DH: Gait asymmetry in patients with limb-length inequality. J Pediatr Orthop,1996.16: 144-50, 16144  1996  [PubMed]
     
    Liu XC; Fabry G; Molenaers G; Lammens J; and Moens P: Kinematic and kinetic asymmetry in patients with leg-length discrepancy. J Pediatr Orthop,1998.18: 187-9, 18187  1998  [PubMed]
     
    Vink P, and Huson A: Lumbar back muscle activity during walking with a leg inequality. Acta Morphol Neerl Scand,1987.25: 261-71, 25261  1987  [PubMed]
     
    Phelps JA, Novacheck TA, Dahl MT. Consequences of leg length inequality in young adults. Read at the Annual East Coast Clinical Gait Laboratory Conference; 1993 May 6; Rochester, MN. 
     
    Richter J: [Differences in the length of legs and walking capacity (a contributionto ergometry in orthopedics)]. Z Orthop Ihre Grenzgeb,1968.104: 548—54, German104548  1968 
     
    Dahl MT: Limb length discrepancy. Pediatr Clin North Am,1996.43: 849—65, 43849  1996  [PubMed]
     
    Etnier JL, and Landers DM: Motor performance and motor learning as a function of age andfitness. Res Q Exerc Sport,1998.69: 136—46, 69136  1998  [PubMed]
     
    Song KM; Halliday SE; and Little DG: The effect of limb-length discrepancy on gait. J Bone Joint Surg Am,1997.79: 1690—8, 791690  1997  [PubMed]
     
    Subotnick SI: The short leg syndrome. J Am Podiatry Assoc,1976.66: 720—3, 66720  1976  [PubMed]
     
    Siffert RS:J Bone Joint Surg Am,1987.69: 1100—6, 691100  1987  [PubMed]
     
    Towheed TE, and Hochberg MC: Health-related quality of life after total hip replacement. Semin Arthritis Rheum,1996.26: 483-91, 26483  1996  [PubMed]
     
    Cauley JA. Epidemiology of total hip replacement. NIH Consensus Development Conference on Total Hip Replacement. Symposium conducted at the National Institutes of Health Annual Conference; 1994 Sept 12; Bethesda, MD. 
     
    O’Brien S; Engela DW; Trainor P; and Beverland DE: Assessing the accuracy of femoral component placement in custom cemented hip replacement. Orthop Nurs. ,1996.15: 47-53, 1547  1996  [PubMed]
     
    Williamson JA, and Reckling FW: Limb length discrepancy and related problems following total hip replacement. Clin Orthop,1978.134: 135-8, 134135  1978  [PubMed]
     
    Love BRT, and Wright K: Leg length discrepancy after total hip joint replacement. In: Proceedings of the Australian Orthopaedic Association. J Bone Joint Surg Br. ,1983.65: 103, 65103  1983 
     
    Jasty M; Webster W; and Harris W: Management of limb length inequality during total hip replacement. Clin Orthop,1996.333: 165-71, 333165  1996  [PubMed]
     
    Abraham WD: Leg length discrepancy in total hip arthroplasty. Orthop Clin North Am,1992.23: 201-9, 23201  1992  [PubMed]
     
    Edeen J; Sharkey PF; and Alexander AH: Clinical significance of leg-length inequality after total hip arthroplasty. Am J Orthop.,1995.24: 347-51, 24347  1995  [PubMed]
     
    Hozack WJ, Lavernia CJ, Ranawat CS. Achieving stability and leg length equality in total hip arthroplasty, acetabular considerations. Instructional Course Lecture at the Annual Meeting of the American Academy of Orthopaedic Surgeons; 2000 March 15-19; Orlando, FL. 
     
    Beattie P; Isaacson K; Riddle DL; and Rothstein JM: Validity of derived measurements of leg-length differences obtained by use of a tape measure. Phys Ther. ,1990.70: 150-7, 70150  1990  [PubMed]
     
    Borg G: Psychophysical bases of perceived exertion. Med Sci Sports Exerc. ,1982.14: 377-81, 14377  1982  [PubMed]
     
    Voorrips LE; Ravelli AC; Dongelmans PC; Deurenberg P; and Van Staveren WA: A physical activity questionnaire for the elderly. Med Sci Sports Exerc,1991.23: 974-9, 23974  1991  [PubMed]
     
    Wilmore JH, Costill DL. Physiology of sport and exercise. 3rd ed. Champaign, IL: Human Kinetics; 1999. p 139. 
     
    Perry J. Gait analysis: normal and pathological function. Thorofare, NJ: Slack; 1992. p 40-1. 
     
    Waters RL, and Mulroy S: Energy expenditure of normal and pathologic gait. Gait Posture,1999.9: 207-31, 9207  1999  [PubMed]
     
    Waters RL; Hislop HJ; Perry J; and Antonelli D: Energetics: application to the study and management of locomotor disabilities. Energy cost of normal and pathologic gait. Orthop Clin North Am,1978.9: 351-6, 9351  1978  [PubMed]
     
    Cander L, Moyer JH. Aging and the lung. Perspectives. New York: Grune and Stratton; 1964. p 132. 
     
    Heyward VH. Advanced fitness assessment and exercise prescription. 2nd ed. Champaign, IL: Human Kinetics; 1991. p 48. 
     
    Lucas C; Stevenson LW; Johnson W; Hartley H; Hamilton MA; Walden J; Lem V; and Eagen-Bengsten E: The 6-min walk and peak oxygen consumption in advanced heart failure: aerobic capacity and survival. Am Heart J,1999.138: 618-24, 138618  1999  [PubMed]
     
    Poole-Wilson PA, Colucci WS, Massie BM, Chatterjee K, Coats AJS. Heart failure: scientific principles and clinical practices. New York: Churchill Livingstone; 1997. p 447. 
     
    Meyer K; Gornandt L; Schwaibold M; Westbrook S; Hajric R; Peters K; Beneke R; Schnellbacher K; and Roskamm H: Predictors of response to exercise training in severe chronic congestive heart failure. Am J Cardiol,1997.80: 56-60, 8056  1997  [PubMed]
     
    Jones NL; Jones G; and Edwards RH: Exercise tolerance in chronic airway obstruction. Am Rev Respir Dis,1971.103: 477-91, 103477  1971  [PubMed]
     
    Ramnemark A; Nyberg L; Borssen B; Olsson T; and Gustafson Y.: Fractures after stroke. Osteoporos Int,1998.8: 92-5, 892  1998  [PubMed]
     
    Costello E; Curtis CL; Sandel IB; and Bassile CC: Exercise prescription for individuals with multiple sclerosis. Neurol Rep,1996.20: 24-30, 2024  1996 
     
    Bassille CC: Guillain-Barré syndrome and exercise guidelines. Neurol Rep,1996.20: 31-6, 2031  1996 
     
    McDonald-Williams MF: Exercise and postpolio syndrome. Neurol Rep,1996.20: 37-44, 2037  1996 
     
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